Win a Free Copy of ANABOLIC PHARMACOLOGY

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  1. Win a Free Copy of ANABOLIC PHARMACOLOGY


    This is your chance to win a free copy of ANABOLIC PHARMACOLOGY. How do you win? Post in this thread the questions that you feel are unanswered or unresolved regarding AAS and other anabolics. I will choose the winner based on what I feel is the best unanswered question (I may choose more than one if I feel the questions warrent it). They do not have to be over-the-top scientific nor do they have to be about AAS -- For example, "Can using insulin to gain muscle result in diabetes?" In fact, they don't even need to be answerable - but plus spare us the absurd.


  2. I feel this is a very under discussed topic on the forums. Most aas talk is geared towards just bbing. What should the aas regimen of a speed athlete, such as a sprinter, look like? How do they cycle for so long, what dosage, etc?
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  3. I wanted to add that subjective questions will not be scored very highly (Questions like "Which AAS is best for bulking") unless they are really unique.

  4. Why do PH/DS give people strange side effects unlike pharmacutical AAS'?

  5. Is there a way to determine what anabolic is best suited for your needs(whatever they might be, BB, Speed, etc) by having blood analized before starting a cycle? Also would you be able to determine what pulsing schedule would work best for you?
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  6. Since we know that the Q-ratio is almost completely useless in determining how an androgen affects a person, is there any other way to predict how an androgen will work in vivo. Maybe looking at the actual molecular structure itself and determining it's affinity for binding to the AR (since flatter structures generally tend to bind to the AR better).

  7. I would like your opinion on the idea of recreational androgen use for skeletal expansion seth: In your opinion could an increase in final adult be achieved by treatment with DHT without letrozole. My understanding of such a thing would be that non aramotizing DHT would cause an increase in bone growth while lowered amount of estrogen due to suppression would in term slow down the process of the growth plate closure. I would be very interested to hear your opinion on such an issue. Thankyou in advance for your time.

  8. if someone has to run a delayed post cycle therapy, what is the best way to dose hcg and clomid? and should they be run together or should the clomid be followed after the hcg?

  9. I have never used a pro-hormone because of the health problems associated with them. High Cholesterol, high blood pressure, androgen suppression, hepatoxicity, etc.

    However, as time goes on and I learn bits more about phs here and there, I am beginning to question the long-term validity of those risks for some products. Suppression can be countered with good pct, cholesterol with other supps and/or diet, etc.

    So, my question is, from a medical standpoint, just how detrimental are Phs (assuming proper on-cycle support and pct) on long-term health? If I were to run a couple of cycles over the next few years, would I be increasing my chances of a heart-attack, stroke, kidney failure, cancer, etc?

    I am trying to look at the big picture here and do not want to jeopardize my well-being down the road for a few pound of LBM now.
    __________________

  10. Good questions so far. Let's keep them coming.

  11. Seth, I dont know if this will be considered a good question or not, it is subjective, but frankly Ive always wanted to ask you. I actually already have your book and its a great great informational guide. Would have been nice to have an index to look like the compound names fast but I cant complain.

    But in your opinion, what are the best say..3 (legal) compounds today and why. It could be any reason, lack of sides compared to results, easy PCT, etc. Not only am I curious myself, but I would love to see your input. I realize thats probably not at all what you were looking for but Id love to see an answer.

    My other question is pulsing, is it really worth it or not?

  12. I was wondering about the level of effect of testosterone usage. Would a cycle be more beneficial for a person with lower natural test levels? Would a person with higher natural levels need to use more for the same effect?

    There seems to be a lot of broscience in this area, but i've never seen anything with actual research.

  13. Based off of the ability to manipulate the chemistry of steroids, and the ability to predict conversion of one hormone into another what can we expect in the future. I mean can we say every steroid ever is already drawn into a book, or based off of the understanding of anabolic androgenic steroids can we expect new creations in the future. As in a another way to tickle our androgen receptors maybe through advancements from current steroids into a chemical compound that can work like a steroids, but have less or side effects. Also how are synthetic steroids created. What raws are used and what methods are used to actually chemically bond every lil element into place to create a large amount of the end product. All a steroid is is simply many smaller pieces put together into something bigger.

  14. Quote Originally Posted by Zero V View Post
    Based off of the ability to manipulate the chemistry of steroids, and the ability to predict conversion of one hormone into another what can we expect in the future. I mean can we say every steroid ever is already drawn into a book, or based off of the understanding of anabolic androgenic steroids can we expect new creations in the future. As in a another way to tickle our androgen receptors maybe through advancements from current steroids into a chemical compound that can work like a steroids, but have less or side effects. Also how are synthetic steroids created. What raws are used and what methods are used to actually chemically bond every lil element into place to create a large amount of the end product. All a steroid is is simply many smaller pieces put together into something bigger.
    Several good questions here. Can't wait to see what else you guys come up with.
  15. UKStrength
    UKStrength's Avatar

    I have one for you Seth regarding PCT and the 'testosterone taper method'.

    It has been discussed quite a great deal on other forums such as t-nation, here's a thread for your convenience (although I'm sure you probably know how it works!)

    Test Taper Protocol

    This method of coming off AAS slowly, as opposed to the sharp halt in androgens following a cycle, is supposedly better for hpta recovery, minimising losses on cycle and reduces the dosages of other PCT agents (e.g. SERMS) during recovery.

    Do you feel this is a viable method for PCT recovery?

    ...and if so could it (and should it) be advised for other compounds such as PH/DS?

  16. if HGH lasts for 1-2 days, and the CJC-1295 version lasts for a week, is there any way to tweak existing compounds to make them last longer? realistically, if we can increase a GH product to last 4x longer, could we do the same to steroids for bi monthly injections?

    myostatin blockers - why isn't this being pursued through all science venues with regards to it slowing down the wasting disease of AIDS?

    cancer cells grow faster when introduced to GH. is it possible to stop the spread of cancer via blocking/shutting down internal GH? follow up question : can we do that while making a strain of GH that is doseable, that cancer doesn't recognize?

    that's all i got right now, sorry if its subpar.

  17. Wouldn't running Clomid 25mgs ED on cycle be a smart way to help prevent the shutdown of natural test production?

  18. Quote Originally Posted by suncloud View Post
    if HGH lasts for 1-2 days, and the CJC-1295 version lasts for a week, is there any way to tweak existing compounds to make them last longer? realistically, if we can increase a GH product to last 4x longer, could we do the same to steroids for bi monthly injections?

    myostatin blockers - why isn't this being pursued through all science venues with regards to it slowing down the wasting disease of AIDS?

    cancer cells grow faster when introduced to GH. is it possible to stop the spread of cancer via blocking/shutting down internal GH? follow up question : can we do that while making a strain of GH that is doseable, that cancer doesn't recognize?

    that's all i got right now, sorry if its subpar.
    There are no subpar questions. Some are more thought-provoking than others but as has been said before, the only bad question is one not asked.

  19. Quote Originally Posted by sethroberts View Post
    There are no subpar questions. Some are more thought-provoking than others but as has been said before, the only bad question is one not asked.
    You know...you could answer all of them

  20. Quote Originally Posted by TexasTitan View Post
    You know...you could answer all of them
    I plan to. Anything that is answered in the book I will tell you it is answered in the book. Anything that is not, I will answer here -- sound fair?

  21. Quote Originally Posted by buquicchioc09 View Post
    I feel this is a very under discussed topic on the forums. Most aas talk is geared towards just bbing. What should the aas regimen of a speed athlete, such as a sprinter, look like? How do they cycle for so long, what dosage, etc?
    I personally don't see much difference except that sprinters would not want to get too bulky. I think a lot of it comes down to diet and some to drug/dose choices. Drugs that cause excessive bloating, liver congestion, and/or joint pain would not be conducive to sprinting - that being said, winstrol seems to be a popular choice even though it can cause joint pain.

  22. Quote Originally Posted by bigmoe65 View Post
    Why do PH/DS give people strange side effects unlike pharmacutical AAS'?
    I personally believe there is a lot of psychosomatic effects being witnessed. Other than that, could you be more specific as to which strange effects are being seen? Lower back pain is seen with DS and AAS, lethargy is seen with both.

  23. Quote Originally Posted by reptone View Post
    Is there a way to determine what anabolic is best suited for your needs(whatever they might be, BB, Speed, etc) by having blood analized before starting a cycle? Also would you be able to determine what pulsing schedule would work best for you?
    I don't believe so, but blood tests may tell you where your SHBG, estradiol, albumin, T3 and test levels are and this may steer you toward the selection of an optimal AAS. Having blood analyzed repeatedl during a pulse would tell you if you need to decrease/increase dose or shorten/extend the period between doses based on the level of suppression thatis seen in the blood tests.

  24. Quote Originally Posted by Schmazz View Post
    Since we know that the Q-ratio is almost completely useless in determining how an androgen affects a person, is there any other way to predict how an androgen will work in vivo. Maybe looking at the actual molecular structure itself and determining it's affinity for binding to the AR (since flatter structures generally tend to bind to the AR better).
    I wouldn't say that the Q-ratio is useless, it just needs to be considered along with other factors such as metabolism, AR binding, PR binding, GR binding, pharmacokinetics, interaction with different enzymes. All of these are potentially in vitro tests that would predict in vivo acivity.

  25. Quote Originally Posted by Random181 View Post
    I would like your opinion on the idea of recreational androgen use for skeletal expansion seth: In your opinion could an increase in final adult be achieved by treatment with DHT without letrozole. My understanding of such a thing would be that non aramotizing DHT would cause an increase in bone growth while lowered amount of estrogen due to suppression would in term slow down the process of the growth plate closure. I would be very interested to hear your opinion on such an issue. Thankyou in advance for your time.
    I think that pharmacological heightening is going on. Doing it as you suggest will result in accelerated linear growth but so would the use of an AI . Probably the best theoretical approach would be to do both to maintain bone density as vertical growth rate is accelerated. It would be a balancing act for sure.
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