Recently i used 6-bromo in my pct, it was in a pct product i was using, and i noticed that my PCT was not going as normal, i followed the same protocol i always use. Here it is....
Week 1 nolva 20mg/ clomid 50mg/ protodioscin 900mg/ cissus drol 2 caps
Week 2-3 nolva 20mg/ clomid 50mg/ protodioscin 900mg/ cissus drol 3 caps
Week 4 nolva 10mg/clomid 25mg/ protodioscin 900mg/ bromocriptine 5mg
Cissus drol 4 caps.
However this is the first time i used 6-bromo all the way through, in normal pct my sex drive goes through the roof, and my strength sky rockets, as my natural test levels peak, this time my libido was terrible, and my strength has dropped off slightly, so i stopped taking the only thing i used that i dont normaly, the 6-bromo in cissus-drol, and what happend, well within 4 days my libido had sky rocketed, and my strength had improved, so to test i took the cissus-drol again.... I started loosing strength, and my libido became non-existent...so here is what i found...
halogens are a row of elements that have very similar chemical behaviour. they inlcude fluorine, chlorine, and bromine
the thing is, 6alpha fluoro testosterone has 1-2 times the anabolic activity of regular testosterone. and 6alpha chloro test has up to 2.8 times the anabolic activity of testosterone. it is expected that 6alpha bromo testosterone (what 6-alpha bromo androstenedione turns into) would be similarly potent as an anabolic hormone. i cannot find data on this compound specifically but i know that in other positions in the steroid, a bromo substitution usually is equivalent to what is seen with a fluoro (both being somewhat weaker than a chloro)
the 6beta fluoro and chloro have only about 20-25% anabolic activity of the 6alphas.
i am reasonably sure that the 6-bromodione avaialble from china is a mixture of alpha and beta isomers. too expensive to seperate
bottom line, there is reason to be concerned about the wisdom of taking a 6-bromo product for PCT. at least one of the isomers likely converts into a potent anabolic steroid. this would make it suppressive enough to render insignificant any HPTA stimulating action acheived via its aromatase inhibitory potential
Your views and thoughts on this would be greatly appreciated, thankyou and kind regards, Russian.
Week 1 nolva 20mg/ clomid 50mg/ protodioscin 900mg/ cissus drol 2 caps
Week 2-3 nolva 20mg/ clomid 50mg/ protodioscin 900mg/ cissus drol 3 caps
Week 4 nolva 10mg/clomid 25mg/ protodioscin 900mg/ bromocriptine 5mg
Cissus drol 4 caps.
However this is the first time i used 6-bromo all the way through, in normal pct my sex drive goes through the roof, and my strength sky rockets, as my natural test levels peak, this time my libido was terrible, and my strength has dropped off slightly, so i stopped taking the only thing i used that i dont normaly, the 6-bromo in cissus-drol, and what happend, well within 4 days my libido had sky rocketed, and my strength had improved, so to test i took the cissus-drol again.... I started loosing strength, and my libido became non-existent...so here is what i found...
halogens are a row of elements that have very similar chemical behaviour. they inlcude fluorine, chlorine, and bromine
the thing is, 6alpha fluoro testosterone has 1-2 times the anabolic activity of regular testosterone. and 6alpha chloro test has up to 2.8 times the anabolic activity of testosterone. it is expected that 6alpha bromo testosterone (what 6-alpha bromo androstenedione turns into) would be similarly potent as an anabolic hormone. i cannot find data on this compound specifically but i know that in other positions in the steroid, a bromo substitution usually is equivalent to what is seen with a fluoro (both being somewhat weaker than a chloro)
the 6beta fluoro and chloro have only about 20-25% anabolic activity of the 6alphas.
i am reasonably sure that the 6-bromodione avaialble from china is a mixture of alpha and beta isomers. too expensive to seperate
bottom line, there is reason to be concerned about the wisdom of taking a 6-bromo product for PCT. at least one of the isomers likely converts into a potent anabolic steroid. this would make it suppressive enough to render insignificant any HPTA stimulating action acheived via its aromatase inhibitory potential
Your views and thoughts on this would be greatly appreciated, thankyou and kind regards, Russian.