Sounds like there might be something to say for it...in theory.
As you may know, the fundamental problem of anti-aging medicine, is how do you counter antagonist pliotropy.
This refers to the fact that throughout most of human evolutionary history humans did not live long full lives. Rather, we tended to be eliminated extrinsically via violence or disease.
For this reason, genes and traits that benefit us earlier in our lifetimes are favored over those that act later.
Natural selection has simply not protected us from aging. How could it? Over millions of years it has acted to promote our welfare while we are reproductive. The further into the reproductive years we get, the less an less we are protected.
It is fundamental that anti-aging specialists understand this very important point.
So now, what of telomeres? I expect their shortening toward senescence is just one of many manifestations of the aging process. But it is very very far from the whole story.
Antagonist pliotropy will act at all genes, on every tissue, so that as we age more and more unselected for complications arise.
This is very important to keep in mind if one wishes to talk seriously about "anti-aging medicine."
