What are the symptoms of low dopamine?
Depression, low sex drive, lack of drive, lack of concentration
True you are correct it could be low T also.
However, most hormones being significantly above or below normal levels can can cause a lot of the same symptoms though really, so I guess the true answer would be to have a full panel blood test done.
yeah my T was low but my prolactin seemed to be okay. but i deffinitly have the symptoms of low dopamine
Depression is more low serotonin, low dopamine is boredom, lack of interest and motivation, poor communication(particurly verbal), lack of confidence.
How do you raise dopamine? Doesdoes the your regulat doc or endo doc check your level of dopamine? Anyone take Ritalian?
Oxitriptan - WorldHealth.net
Posted in Hormones & Pharmacological Agents on Wed March 19, 2008
Oxitriptan or 5-hydroxy-tryptophan (5HT) is converted into the neurotransmitter serotonin in the body. Serotonin is a key factor in mood regulation, and the reduction of depression and anxiety. A lack of serotonin has been linked to compulsive disorders, especially the overeating of carbohydrates. Serotonin is also the precursor to the pineal gland’s production of melatonin. As serotonin levels decline with age, supplementation with the amino acid L-tryptophan (the body produces serotonin by converting L-tryptophan to 5 hydroxy-tryptophan via a vitamin B3 dependent enzyme, and then converting 5HT to serotonin via a vitamin B6 dependent enzyme), or the drug Oxitriptan is beneficial. Oxitriptan’s advantage is that, unlike L-tryptophan, it is used solely by the brain for the manufacture of serotonin, and is thus more effective in increasing serotonin levels.
ROLE FOR ANTI-AGING:
Oxitriptan’s anti-aging benefits may include the prevention and treatment of depression, maintenance of serotonin levels as the body ages, prevention and treatment of compulsive disorders like overeating, improved daytime alertness and treatment of insomnia.
So how do you raise dopamine then?
I'm on an amino acid program to balance neurotransmitters. Amino acids can cross the blood-brain barrier and raise neurotransmitter levels in the central nervous system. L-tyrosine is the amino acid that raises dopamine. So does mucuna pruriens. You have to have a 'tyrosine base' before adding mucuna. Try working up to 1 - 1.5 grams of tyrosine 3 times per day on an empty stomach. Then add 300 mg mucuna to each of the 3 tyrosine doses.
i am currently on some adrenal fatigue meds and my testosterone is normal about 600 and i can have sex, although some more motivation wouldn't hurt
in the past i used to take a product called dopafibra which has 400 mg mucuna pruriens and it made me feel a lot better
also tyrosine did this
how do i know it's not just the stimulating function of tyrosine that's at work, but that it in fact is increasing dopamine?
may be my final missing link
I've spoken to a Dr on another board, and he told me to stay away from l-tyrosine and stick only to l-dopa (because of adrenals).
fwiw, I have been using Unique Nutrition's L-dopa (not mucuna puriens). It's not stimulating, it tends to make me a bit sleepy.
L-DOPA (3,4-dihydroxy-L-phenylalanine) is a naturally occurring amino acid found in the human brain and in certain plants (Macuna pruriens aka velvet bean). L-Dopa is produced from the amino acid L-Tyrosine via the enzyme tyrosine 3-monooxygenase (previously called tyrosine hydroxylase). Afterwards L-Dopa is able to cross the blood-brain-barrier and converts into Dopamine by the Vitamin B-6 dependant enzyme aromatic-L-amino-acid decarboxylase.
Something else to keep in mind:
Conversion to dopamine also occurs in the peripheral tissues, i.e. outside the brain. This is the primary mechanism of the adverse effects of levodopa. It is standard clinical practice to co-administer a peripheral DOPA decarboxylase inhibitor—carbidopaCarbidopa
Carbidopa is a drug given to people with Parkinson's disease in order to inhibit peripheral metabolism of levodopa....
Benserazide is an inhibitor of DOPA decarboxylase that does not enter the central nervous system....
—and often a catechol-O-methyl transferaseCatechol-O-methyl transferase
Catechol-O-methyl transferase is an enzyme first discovered by biochemist Julius Axelrod....
(COMT) inhibitor, to prevent synthesis of dopamine in peripheral tissue. Co-administration of pyridoxine without a decarboxylase inhibitor accelerates the extracerebral decarboxylation to such an extent that it cancels out the effects of levodopa administration, a circumstance which historically caused great confusion.