[idunk42]

Originally Posted by LakeMountD

It isn't needed though just so you realize that.

I understand that Lake, I just do it from potentially wasting a little bit of the igf, plus i feel gipt only injecting that little of substance.

[LakeMountD]

Originally Posted by idunk42

I understand that Lake, I just do it from potentially wasting a little bit of the igf, plus i feel gipt only injecting that little of substance.

Haha easy killer that was written towards him not you . I know you know that, I just didn't want him to think he couldn't use his IGF at all if he didn't have it.

[idunk42]

Originally Posted by LakeMountD

Haha easy killer that was written towards him not you . I know you know that, I just didn't want him to think he couldn't use his IGF at all if he didn't have it.

LOL.....ok my bad. Today's and off day, so i got some built up energy and emotion.

[preston25]

Just started IGF-1 would like to stack with aas for strength. thinking of dianabol. Is it best to take at the same time or in between cycles. Im an undurance athlete so my cycle is for strength only.

Best
Preston25

[idunk42]

Originally Posted by preston25

Just started IGF-1 would like to stack with aas for strength. thinking of dianabol. Is it best to take at the same time or in between cycles. Im an undurance athlete so my cycle is for strength only.

Best
Preston25

Well you probably dont wanna run dbol without any test. Also, if your an endurance athlete, Im not sure if dbol is what your looking for. It has a tendency to make you hold quite a bit of water. Do some more reading on this board about AAS in general, to help you get a better grasp of the subject.

[LakeMountD]

And why are you going for strength as an endurance athlete? It is sort of two different muscle fibers completely.

IGF works great in synergy with AAS and during PCT, whichever you prefer. In theory it is best if used with AAS since the increased anabolism could increase hyperplasia more so than without it.

[drveejay11]

Originally Posted by basskiller

Very nice LMD

You may want to have your study listed like this.
It's very much easier for people to see what works best throughout your member study..
Again, Really Nice!!!



Members were asked:
1. How many cycles have you done?
JBlaze: Only one so far. Did it during PCT. My next cycle will be during an AAS cycle.
ManBeast: One cycle of IGF1-LR3 during PCT.
Nuteboy: How many cycles have you done? One cycle.
Longdog: 2 cycles of IGF1-LR3. 1 off cycle & 1 during PCT.
BryanFury: One stand alone cycle
er700: 1 cycle along with test and eq
IntResearch: 2
Raprazant: 1

2. How long were you on igf-1?
JBlaze: 21 days, at this point i noticed the gains stopped coming, so i decided to come off.
ManBeast: 28 days.
Nuteboy: Five weeks.
Longdog: 4 weeks & 3 weeks
BryanFury: 22 days
er700: 25 days
IntResearch: 1 month
Raprazant: 30 days

3. How many mcg did you use?
JBlaze: I used 40mcg in the beginning and bumped it up to 60 mcg. At 60mcg i noticed better results, so my next cycle will consist of 60mcg throughout.
ManBeast: 30mcg ED
Nuteboy: First three days I used 200mcg per day but once I was told how to calculate mcg's I'd do 40mcg per day.
Longdog: 30mcg ED
BryanFury: 40mcg ED
er700: 40
IntResearch: 40/75
Raprazant: 100 mcg/day with one day off every 6 days

4. How many times a day did you shoot?
JBlaze: Once a day after my workout, and first thing in the morning on non-workout days.
ManBeast: Once a day, sometime after my workout.
Nuteboy:.Once per day. Usually after my workout.
Longdog: Once a day, IM bilaterally into muscle worked, immediately after training.
BryanFury: Once a day, pre-workout
er700: once a day after workouts
IntResearch: Once
Raprazant: once, after workouts

5. How much fat loss and muscle gain was there overall?
JBlaze: Gained 4lbs of muscle and lost 1.5lbs of fat. Keep in mind this cycle was during PCT.
ManBeast: Lost some fat and gained definition during PCT.
Nuteboy: How much fat loss and muscle gain was there overall? Difficult to figure how much fat lost but I am leaner this offseason than last year.
The first four days I gained 4 to 5lbs. Overall I'd say 8lbs of solid muscle.
Longdog: Once a day, IM bilaterally into muscle worked, immediately after training.
BryanFury: Lost some fat, body comp. changed overall. Gained roughly 5lbs.
er700: ~2 pounds of muscle and loss ~ 3 pounds of fat. At the time I was getting ready for a powerlifting comp. and was eating very clean to get my bwt. down to 220, I normally weigh over 240.
IntResearch: 2 loss, 5 gain
Raprazant: 6 lbs. of fat loss, 4 lbs of muscle gain give or take a lb of each

6. How likely are you to use it again?
JBlaze: I already have 2 bottles sitting here, and i got 2 more on the way. I'll never do PCT w/o this again.
ManBeast: Very likely.
Nuteboy: Very. I will use it again "offseason" but will include insulin.
Longdog: Will definitely use it again, but will use 40mcg or more.
BryanFury: Plan on a second 4 weeks after the last ended.
er700: Definitely, getting ready to use it along with my PCT
IntResearch: very likely, only dislike was getting sleepy after injecting
Raprazant: very likely but with hgh

7. What strength gains did you see?
JBlaze: Hardly any at all, but then i do extremely slow concentrated movements, i rarely go up in weight.
ManBeast: I don't attribute the strength gains made to this, more to the low-rep lifting scheme I was using.
Nuteboy: Not much in the strength gains as I normally lift heavy.
I would not say IGF made me stronger but made me look FULL as hell.
Longdog: No strength gains. Look elsewhere if that's what you want, this is not an androgen.
BryanFury: Strength was placebo effect I think. Muscles did get more full with increased vascularity.

8. Finally did you use AAS with the IGF -1?
JBlaze: This cycle it was igf-1 alone, but my next cycle i will be using it week 5-9 of my AAS cycle. Then my 3rd and final cycle for a while will be during my last 2 weeks on AAS, and 2 weeks into PCT.
ManBeast: Nope
Nuteboy: Yes. I used Test and Deca.
Longdog: No, but I will use it on a cycle next time.
BryanFury: No

Worthy of a bump. Excellent advice.

[preston25]

Thanks for the reply, im looking for something to cycle in with my igf-1. aas. something that doesnt create water gain, but increases strength. i welcome ideas but will keep researching.

preston25

[xtraflossy]

Originally Posted by LakeMountD

Sheesh I got completely shut out of that one hahaha.

Thought you could use a break!!!

[joebig]

Is the MGF by IBE short acting or the LR3?

[LakeMountD]

Originally Posted by joebig

Is the MGF by IBE short acting or the LR3?

There is no such thing as LR3 MGF. There is, however, LR3 IGF-1, which is longer acting then rhIGF-1, which is seen in the medical field.

There is a longer version of MGF coming out too, though. It is the PEGylated version.

[BassD]

@LakeMountD

I quote what you posted recently on b o d y b u i l d i n g . c o m :

The most recent update was the biggest one and if you haven't downloaded it recently, definitely go get the new one. It was recently found that IGF-1Ea is the actual activation mechanism of stem cells and not MGF as in Dr. Goldspink's older results. That is a huge discovery by the way and is explained in the article.

Do you have any additional info on this at this time??

[LakeMountD]

Originally Posted by BassD

@LakeMountD

I quote what you posted recently on b o d y b u i l d i n g . c o m :




Do you have any additional info on this at this time??

Still the same info. The problem occurred in Dr. Goldspink's older studies. When he first began research on MGF he hypothesized that it was responsible for not only proliferation (brining in of myoblasts) but also for differentiation (activation of these myoblasts). This, however, proved to not be true in newer studies and actually it was wrong to a very large degree as it was found that not only did MGF not differentiate myoblasts but it actually inhibited myoblasts from differentiating (this is his newest hypothesis). They now say that IGF-1Ea is responsible for the differentiation, which is good for all the LR3 IGF-1 users out there. MGF still has its place as something that has a lot of potential, especially with the longer lasting PEGylated version coming out, but it will take much longer to figure out how to use it synergistically with LR3 IGF-1 since it does inhibit differentiation.

We basically have to determine how we can dose this stuff to where we get a large influx of myoblasts without inhibiting differentiation. I also still believe LR3 IGF-1 and MGF stacked together are the best way to go. With the large amount of IGF-1 we are getting, we need the extra myoblasts and with all the myoblasts being proliferated by the MGF, you are going to want the added anabolism.

[BassD]

Originally Posted by LakeMountD

Still the same info. The problem occurred in Dr. Goldspink's older studies. When he first began research on MGF he hypothesized that it was responsible for not only proliferation (brining in of myoblasts) but also for differentiation (activation of these myoblasts). This, however, proved to not be true in newer studies and actually it was wrong to a very large degree as it was found that not only did MGF not differentiate myoblasts but it actually inhibited myoblasts from differentiating (this is his newest hypothesis). They now say that IGF-1Ea is responsible for the differentiation, which is good for all the LR3 IGF-1 users out there. MGF still has its place as something that has a lot of potential, especially with the longer lasting PEGylated version coming out, but it will take much longer to figure out how to use it synergistically with LR3 IGF-1 since it does inhibit differentiation.

We basically have to determine how we can dose this stuff to where we get a large influx of myoblasts without inhibiting differentiation. I also still believe LR3 IGF-1 and MGF stacked together are the best way to go. With the large amount of IGF-1 we are getting, we need the extra myoblasts and with all the myoblasts being proliferated by the MGF, you are going to want the added anabolism.

With this in mind, what do you think of adding some Arachidonic acid (key ingredient in "X-Factor") into a stack of MGF and LR3 IGF-1 ??

Arachidonic acid intensifies IGF-1 signaling, and supports muscle hypertrophy by increasing satellite cell (myoblast) fusion in muscle fibers.

Or maybe an cheaper option.... replacing the LR3 IGF-1 with Arachidonic acid ??