Ketogenic and blood sugar

  1. Ketogenic and blood sugar

    One difference between fasting and a ketogenic diet is that the slight insulin response to dietary protein will cause blood glucose to be maintained at a slightly higher level, approximately 80-85 mg/dl.  This most likely occurs due to the conversion of dietary protein to glucose in the liver.

    (Mcdonald, The Ketogenic Diet)

    Has anyone had experience with having low blood sugar while on a ketogenic diet?  I tested using my friends diabetic supplies and came up at 60.  I could definitely tell that I was low- had a spaced out feeling, slightly cold, and shaking a little bit. 

    Can someone more knowledgable explain how the consumption of dietary fat, gluconeogensis, and glycogen levels plays a role in blood sugar levels while in ketosis?



  2. I know the addition of dietary fat reduced the amount protein is converted into glucose. If your fat consumption is high enough gluconeogensis shouldn't occur that often. That usually happens when you glucose and calorie levels are extremely low, or in times of extreme stress (post surgery, etc...) If you take in just protein (whey shake) and your fat intake isn't high enough, most of they whey will get converted in glucose, hence the insulin spike. THats why its recommened to eat more whole foods and less shakes while doig a keto. I never have a shake wioth just protein. I still use shakes but always add a good amount of fat to it (canola, flax, hemp, etc...)

    Forget to add, fat has been determined to cause a significant release of insulin than previously thought and the new II is supposed to measure the glycaemic load of particular foods. So in fac the transport of nutrirnets still occurs at a relatively high rate without the glucose spike.
    For answers to board issues, read the Suggestion and News forum at the bottom of the main page.

  3. From the man himself:

    "Glucagon, insulin and ketogenesis:
    The formation of KB's and utilization of fuel is ultimately controlled by
    the circulating levels of insulin and glucagon. Insulin is a hormone
    released from the pancreas in response to eating carbohydrates. Glucagon
    is insulin's antagonistic hormone and is only present when insulin levels
    fall to quite low levels. In the liver, high glucagon levels direct FFA
    away from TG synthesis and towards beta-oxidation. Glucagon also activates
    adipose tissue lipase which activates lipolysis. Glucagon's ketogenic and
    lipolytic effects are inactivated by even small amounts of insulin. To
    achieve sufficient glucagon concentrations for increased
    ketogenesis/lipolysis, blood glucose levels must drop to around 50-60 mg/dl
    and insulin must drop almost to zero. This drop in insulin can occur with
    complete fasting, exercise, or by simply restricting carbohydrate intake to
    below 30 grams per day. Within about 3 days of carbohydrate restriction,
    blood glucose will fall below 60 mg/dl, insulin levels will drop to zero
    and glucagon levels will increase causing an increase in KB formation.
    With exercise training, ketogenesis should occur more quickly and ketosis
    established. (2)

    Establishment of ketosis, even in the short term, has the effect of
    increasing the body's ability to utilize fat for fuel. After adaptation to
    ketosis, there is a decrease in fasting RQ (an indicator of relative fuel
    metabolism with lower values indication greater reliance on fat metabolism
    versus carbohydrate metabolism) (7). Also, there is a decrease in glucose
    oxidation during ketogenic diets as KB's are providing much of the body's
    energy needs (18). Additionally, adaptation to a ketogenic diet increases
    fat oxidation during exercise even in trained individuals (14, 17).

    One point of contention regarding ketogenic diets is the supposed protein
    sparing effect when compared to a eucaloric diet with a high carbohydrate
    intake. Due to methodological differences, some studies have found a
    decrease in protein utilization while others have found an increase (8).
    However, available data seems to support the idea that ketosis spares
    protein from being used for energy. Since there is essentially an
    unlimited supply of fat which can be converted to ketones, and since
    ketones can be used by all oxidative tissues, there should be little need
    to oxidize protein to generate glucose through gluconeogenesis. There is
    an obligatory protein requirement which must be met of about 30 grams per
    day. And, to be safe, an intake of 60-75 grams of protein is recommended

    Other effects of low carbohydrate diets
    Additionally, a low carbohydrate intake will allow for overall greater
    lipolysis and free glycerol release when compared to either high
    carbohydrate or normal diets (7, 12). This is mediated in part by the lack
    of insulin, which has a lipolysis blocking action even at low
    concentrations as well as increases in other lipolytic hormones such as
    growth hormone, glucagon, the catecholamines, and glucocorticoids. (7)
    Additionally, growth hormone levels increase on low carb diets which will
    further help to prevent the inevitable protein losses which occur when
    calories are restricted (2, 7).

    This help? I assumed you had this but you never know
    For answers to board issues, read the Suggestion and News forum at the bottom of the main page.

  4. Thanks bro...


  5. Just watch your blood sugar wyd don't let it fall to low. I would rather see it high than deathly low. Keep it going and lose the weight.



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