judge-mental
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Can we structure around Long Ester peaks and decline adjusting amount of short acting?
for instance:
If Test E:
Peak in blood appears after 12-15 hours and this is shortened to 8-11 hours to the 16th injection(1) (this is probably to being shut down and HL being a little longer than 1 week)
HL is 145-177h (6-8) days
so in essence if one is injecting every week, one could stack orals like so:
day 1 : inject x mg Test E
day 2 :nothing
day 3: inject/use oral steroid with short HL at low dose
day 4: same as day 3
day 5: inject/use oral steroid with short HL at medium dose
day 6: inject/use oral steroid with short HL at med-high dose
day 7: inject/use oral steroid with short HL at high dose
Repeat
one can use acute effects of said peaks/short acting AAS for workout boosts, while more importantly, keep androgenic and anabolic signals constant. one the things IMO and IME that really suck about long esters is up and down of mood, immune, etc. considering the graphs its obvious why! the consequences on long term hypothalamic regulations are not pretty, stabilizing with inverted curve short acting aas mya be a crude but effective mechanism to do this ( I know people have been doing this in the end of cycles, but why not from the beginning?)
Thoughts? or is this really delusional?
for instance:
If Test E:
Peak in blood appears after 12-15 hours and this is shortened to 8-11 hours to the 16th injection(1) (this is probably to being shut down and HL being a little longer than 1 week)
HL is 145-177h (6-8) days
so in essence if one is injecting every week, one could stack orals like so:
day 1 : inject x mg Test E
day 2 :nothing
day 3: inject/use oral steroid with short HL at low dose
day 4: same as day 3
day 5: inject/use oral steroid with short HL at medium dose
day 6: inject/use oral steroid with short HL at med-high dose
day 7: inject/use oral steroid with short HL at high dose
Repeat
one can use acute effects of said peaks/short acting AAS for workout boosts, while more importantly, keep androgenic and anabolic signals constant. one the things IMO and IME that really suck about long esters is up and down of mood, immune, etc. considering the graphs its obvious why! the consequences on long term hypothalamic regulations are not pretty, stabilizing with inverted curve short acting aas mya be a crude but effective mechanism to do this ( I know people have been doing this in the end of cycles, but why not from the beginning?)
Thoughts? or is this really delusional?
