I sent that same question to ALRI and have not got a response. After thinking about this I have come to the conclusion that is not a good idea.
This is from the write up
"Everyone has heard of the powerful thyroid hormone T-3. And of course many have reported the benefits from the T-3 analog Triac. Triprop is a naturally occurring thyroid analog of T-3 similar to Triac in some positive regards".
Just based on that
It would be equilvalnt to taking tri max for T3 pct. Taking another thyroid hormone after being on T3.
Someone will have to get bloodwork to see the real effect. I'm betting thyrogen will suppress thyroid production post cycle. I love ALRI and expect there products to be potent but there ad's are a little inflated.(max-lmg has no effect on libidio) yeah right
Will do, if I get a response. it is interesting to note in there write up about the stuff they use to inhibit the negative fedback loop(tyrosine ester and some other ****). If that had any chance of working at all I wouldn't mind getting that as a seprate product and then a person lcould run that with a T3 cycle.
This is from there website Invalid Link Removed
THYROID FEED-BACK MECHANISMS and FEED-BACK LOOPS
Of course the thyroid does not simply mass produce thyroid hormones continuously. Actually it tries pretty hard to produce as little as possible. This is due to the "checks and balances" nature included in the action/reaction factors for the endocrine system called "feedback-mechanisms". In the case of thyroid function, the feedback-mechanism or loop involves the hypothalamus (secretes TRH), pituitary gland (secretes TSH), thyroid gland (secretes T-4), and the liver (converts T-4 into T-3). A feedback-mechanism or loop can trigger the release of another hormone (positive feed-back), or inhibit its release (negative feed-back) thus maintaining that balance. This means high levels of T-4 or T-3 initiate a negative feed-back loop that tells the hypothalamus to produce less TRH, and low levels of T-4 or T-3 initiate a positive feed-back loop that tells the hypothalamus to produce more TRH. So, simply supplementing with higher than normal levels of thyroid hormone analogs will result in a negative feed-back loop that tells the pituitary to stop producing TRH and the hypothalamus to get greedy with TSH…and natural thyroid hormone production goes in the toilet with you metabolism. Hmmm, picture what happens when you stop taking the extra thyroid analogs. The obvious effective goal therefore becomes to maximize total circulating thyroid hormone levels both supplementally…and provide synergistic support to natural thyroid hormone production by our bodies. One or the other simply is inefficient. So now that we have the powerful ALRI analogs Triprop and Diprop to add to total circulating levels, we need to inhibit the negative feed back-loop.
Natural Thyroid Function Support
Thyroid hormone production in the body begins with the amino acid tyrosine. Unfortunately free-form tyrosine really does not pass well into the circulatory system at a very high rate orally. Like most amino acids (but not all) a lipophylic carrier or such as an ethyl ester has the potential to increase oral bioavailability up to 400%...so yes, we created tyrosine ethyl ester for Thyrogen-X™. Now that we have the raw material for natural thyroid hormone production, we need to maintain or even increase the natural production signals by triggering TRH and TSH release from the pituitary and hypothalamus. In rat studies freeze dried extract of Olea europaea (olive leaf extract) was shown to have a maintained stimulatory affect upon the pituitary-thyroid axis that resulted in increased thyroid gland activity. The study shows that this is not a result of pituitary stimulation but of hypothalamic stimulation thus supporting TSH activity. Which is pretty cool, huh? Clary sage is a rather interesting herbal goodie that can be standardized for many of its active compounds. In one study it was shown that, as an aroma therapy, it aids in improving stress scores significantly. Okay, but the really cool effect it has is due to its cAMP stimulating value. There is a significant similarity between compounds in this interesting herb and Thyroid Stimulating Hormone (TSH). As most are aware, increased TSH means more fat burning anabolism promoting T-3 production (yes, it acts synergistically with the next goodie in this regard…but through different pathways). The connection here is that the TSH-like effect triggers an enzyme called adenylate cyclase in the thyroid that results in an increase in cAMP activity and subsequently lots of extra T-3 production…naturally. Since there is an increase in cAMP, there is an accompanying increase in nitrogen retention. There appears to be some research that supports the belief that increased cAMP results in greater LH release as well (you know, that stuff that tells “The Boy’s” to make more testosterone? Okay, the LH part affects only the guys, women don’t have testes…or shouldn’t). Think about the HPTA stimulating synergy between Clary sage extract and thyroid hormones explained prior. Hmmmm? Beta AET ECPE (beta-androstenetriol ECPE)? We have all heard, and in many cases experienced, the positive effects of DHEA and its even better metabolites. As example are the patented and effective products 7-OXO-DHEA and of course 7-Hydroxy-DHEA analogs. They are noted for their unique ability to avoid conversion into androgenic metabolites or affect androgen receptors while promoting fat loss, lean mass retention and even maximizing thyroid gland activity. Of course oral bio-availability is pretty poor with most of these analogs thus requiring higher dosages. Did I mention that bAET (b-androstenetriol) is between 100 and 100,000 times more active than its DHEA precursor metabolites? Okay, how about that bAET ECPE is nearly 100% orally bioavailable? Yup. It’s why we formulated it. Okay, let me explain a little about the fat loss and lean muscle side of things in regard to bAET ECPE…
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