There are no studies I am aware of that indicate that oral Ecklonia cava extract will effect DHT.
There are some that indicate that if you soak hair follicles and other things in Dieckol it may effect DHT levels in the sample. Its not logical to imply that oral EC would have the same effects.
https://www.ncbi.nlm.nih.gov/pubmed/22754373
The study that was referenced above used hair follicles soaked in 35% dieckol=
"When vibrissa follicles were cultured in the presence of E. cava enzymatic extract (which contains more than 35% of dieckol) for 21 days, "
EC(ecklonia cava) extract was tested Vs viagra and beat viagra on most tests.
The vast majority of users mention an increase in sexual function.
Also....Not all EC extracts are the same.
Some standardize for seanol, some for dieckol, some are full spectrum. And some are very weak while some are very strong.
Ours is exceptionally strong.
The point is you cant say 1000mg of ... ec product is comparable to ... ec product without knowing how they were extracted, strength, standardization etc.
By the way the strongest EC extract I know of is in FOLLIDRONE 2.0
More info regarding EC and sexual function.
=======================
ERECTILE FUNCTION - ECE V. VIAGRAÆ
Nitric Oxide
ECE can regenerate the vascular endothelium, the cells critical to the inner lining of the blood vessels. They generate the chemical nitric oxide (NO), which keeps the arterial walls relaxed and dilated. After a six-week study of ECE, flow mediated dilation and NO mediated dilation increased by 60% and 50%. In another study, coronary artery disease patients were given ECE for six weeks. Blood flow controlled by NO increased 50-60%. These results confirm that ECE can rejuvenate damaged endothelial cells to produce NO. This effect was further confirmed in a study on erectile dysfunction (see below). Interestingly, ViagraÆ works by increasing NO in the penile artery.
ECE v.ViagraÆ: 8-Week Clinical Trial
Scientists studied 31 men with erectile dysfunction (ED) for over six months. They compared eight weeks of ECE use to ViagraÆ. They looked at orgasmic function (OF), intercourse satisfaction (IS), overall satisfaction (OS), and erectile function (EF). Over those eight weeks, ECE scored 87%, 74%, 62%, and 66%. ViagraÆ scored 27%, 44%, 39%, and 66%. No side effects were reported with ECE:
Population with 25+% Improvement in IIEF (International Index of Erectile Function) score was as high as 81%. Total IIEF score significantly increased from 29.1 ± 13.1 to 47.0 ± 14.5 with 62% of improvement. When the IIEF scores were grouped into five separate domains, mean IIEF scores at the 8th week were significantly greater than those at week 0 for all domains (all p<0.01). The degree of improvement was significant in the following order: OF (87%), IS (74%), EF (66%), and OS (62%). Scores on key questions (asking frequency of penetration and asking frequency of maintaining an erection after penetration), which directly indicate the ability to achieve and maintain an erection sufficient for sexual activity, were improved up to 74% and 77%, respectively (p<0.01).
It is very important to note that despite the marginal improvement in sexual desire (20%) that is of psychological nature, great improvements were reported in the domains directly related with erection that is of physical nature and dependent on normal vascular function of the penile artery.
Also noteworthy, was a significant increase in the orgasmic function score (87%), intercourse satisfaction (74%) and overall satisfaction (62%) as well as erectile function (66%) in comparison with the results for sildenafil reported by Marks, et al. (Marks, et al., 1999) (27%, 44%, 39% and 66%, respectively), which indicates that ECE significantly contributed to the normalization of the general vascular conditions around the sexual organ.
These results strongly indicate that the long-term administration of ECE significantly contributes to the neutralization of oxidative risk factors, thereby improving peripheral blood circulation around muscles and nerves involved in sexual function as well as the penile artery. No side effects were reported.
Vasodilation & Erectile Function
It has been reported that vasculogenic ED patients have elevated levels of angiotensin II for the duration of the erection process. The demonstrated action of ECE on ACE and resulting vasodilation is thought to play an important role in inducing successful erectile function.
Long-Term Improvement Via Vascular Protection
As discussed, ECE phlorotanins have potent antioxidant and anti-inflammatory effects. Together with ECE's ACE inhibitory activity, which is also beneficial to vascular homeostasis, these activities, upon long-term oral administration, may all contribute to supporting a healthy vascular system, including the penile artery.
