Question about Maca and Libido
- 09-21-2009, 08:34 PM
Question about Maca and Libido
Hey guys! Long time since I've been on the forum.
Basically I took some stuff I shouldn't have a long time ago and basically messed up my libido/sex drive. Getting a full firm erection can be difficult sometimes.
Since I have stopped taking any form of prohormones, I have noticed slow (and I mean slow) improvement in sex drive. It has been getting better over time and my endocrinilogist has told me it will probably take a few months till it recovers. Also since Ive been off, the small bit of Gyno I had has been decreasing in size
Anyhow! My main question is that I've been using this vitamin product I got from GNC called Maca Man. I believe it contains mainly Yohimbe and Maca. Anyways, it works great for me. It enhances my libido perfectly and I love using it.
Is this ok to use during my "time of recovery" from prohormones? It seems like a great fix for me to use during this time while my body recovers, but I'm not sure if it is throwing off the recovery or not.
Any comments or help is greatly appreciated
- 09-22-2009, 10:53 AM
I doubt it is throwing off the recovery, it shouldnt suppress you in anyway that I am aware of, someone correct me if I am wrong. Also you may want to look into something like Anabolic Xtremes Advanced PCT, that should help you bring your guys back sooner.
09-22-2009, 12:53 PM
No problem in using it, but you could do way better than that product. I guarantee you the maca is underdosed.
A number of products would do you better: diesel test hardcore, san endotest pro, testopro, blue gene, etc.
09-22-2009, 02:34 PM
09-22-2009, 04:15 PM
Ok, thanks guys appreciate the comments huge. You're much more helpful then bodybuilding . com thats for sure.
Now Ive been told by the doctor as well as somebody builders to avoid any type of PCT or testboosters to let my body regulate naturally on its own. Naturally, how long does this process take? My endocronologist said a couple months, but I dont really take what she said seriously because she didnt even know what a prohormone was :S
I miss my full blown sex drives and random erections hahaa
09-25-2009, 02:30 PM
Using maca thats sort of thing doesn't appear to boost test directly. It does help with the rest of the system and may have neuro-steriodal effects. I'd use DTHC to ramp your overall well being. I would say maca/trib/long jack/icariin/yohimbine hcl will all help but there is no evidence of directly boosting test at all. There are anecdoctal reports but nothing concrete. I vouche that icariin is 'amazing'. You can also look to use divanil products.
Anyway maca is cheap so if it works you can run it for a long time.
The real question is whether to use an AI, clearly your endo says no. The board here will say yes.
The endo's problem is that there is nothing they can prescribe because exogenase hormone would be counterproductive, that is very different from saying nothing will work.
Pharmacology began in herbalism and IMO the more I know the more I think herbalism is the way to go for anything but moderate to serious medical conditions. In your case this ain't that serious, just stay off PH if its causing so many problems PCT.
BTW happened to your PCT, it looks like you didn't have one in place.
09-25-2009, 11:38 PM
Ive done quite a bangup job on my body Ive realized as my libido is still low (I think its been improving though), and Ive learned my liver is pretty damaged (I find out on Monday what the exact problem is) :S
Because of the damage to my liver, I am not putting anything pill form in my body at this time to let it repair itself. Hopefully my libido and test levels will rise to normal soon. Ive been told my test levels are normal, but still on the poor side. 11.4 free test, and 340 total test.
09-26-2009, 03:36 PM
Once you see your liver values, which can't figure out why endos don't just prescribe a 3-4 week clomid taper for you fellas that are having a difficult time recovering.
SD is the only one that crushes production that harshly. I've ran something like 6 legal designers and I'd probably run 5 of them again when weighing pros vs. cons. Obviously SD is not in my future plans again LOL
09-26-2009, 05:04 PM
With that being said, what kind of recovery time do you think I can expect before my libido is full kicking again?
09-28-2009, 12:25 PM
With proper pct, I felt like garbage for a good 3 weeks. That's when I was like "ok things are getting better" in terms of libido.
I'd say it was about 3 months before everything seemed like it felt completely right again (libido, mood, sleep patterns).
When I eventually got bloodwork, everything was in check except for HDL. That took the longest to resolve.
09-28-2009, 12:37 PM
09-28-2009, 03:42 PM
Ya I had Torem in pct. I suspect the Torem actually made HDL worse, even though it's the serm that is supposed to be HDL friendly.
HDL, in short, is "good" cholesterol. The higher the better. I'm thinking like 50ish is pretty normal and mine was 30ish IIRC with the bloodwork. I was back to normal like a year later... that's the next time I had bloodwork so I dunno actually how long it took to get back to proper levels.
09-30-2009, 04:55 AM
09-30-2009, 07:51 AM
09-30-2009, 08:18 AM
09-30-2009, 05:20 PM
10-02-2009, 03:48 AM
Toremifene (Fareston) lowered LDL (bad Cholesterol) and increased HDL (good cholesterol) when given to men receiving androgen-deprivation therapy for prostate cancer.
Study published in Journal of Clinical Oncology, April 2008
Massachusetts General Hospital Cancer CenterWe evaluated the effects of toremifene, a selective estrogen-receptor modulator, on fasting serum lipid levels in men receiving ADT for prostate cancer. PATIENTS AND METHODS: In an ongoing, multicenter, double-blind, placebo-controlled phase III fracture-prevention study, 1,389 men receiving ADT for prostate cancer were randomly assigned to receive toremifene (80 mg/d) or placebo. In this interim analysis of 188 patients, changes in fasting serum lipids from baseline to month 12 were compared between the placebo and toremifene groups. RESULTS: Changes in serum lipids differed significantly between the groups. Mean (+/- SE) total cholesterol decreased by 1.0% +/- 1.7% from baseline to month 12 in the placebo group and decreased by 8.1% +/- 1.4% in the toremifene group (P = .001 for between group comparison). Low-density lipoprotein (LDL) cholesterol increased by 0.8% +/- 2.5% in the placebo group and decreased by 8.2% +/- 2.5% in the toremifene group (P = .003). In contrast, high-density lipoprotein (HDL) cholesterol decreased by 4.9% +/- 1.2% in the placebo group and increased by 0.5% +/- 2.2% in the toremifene group (P = .018). Triglycerides increased by 6.9% +/- 4.2% in the placebo group and decreased by 13.2% +/- 3.6% in the toremifene group (P = .003). CONCLUSION: Toremifene significantly decreased total cholesterol, LDL cholesterol, and triglycerides, and increased HDL cholesterol in men receiving ADT for prostate cancer.etc etcThe objective of this study was to evaluate the different profiles of serum lipids resulting from the administration of selective estrogen receptor modulators (SERMs). Postmenopausal primary breast cancer patients (n = 197) with node-negative, hormone receptor-positive who were treated at our department or in other related medical institutions from April 1997 through March 2001 were given adjuvant therapy. The adjuvant therapy included 1 year's administration of tamoxifen (TAM) 20 mg or toremifene (TOR) 40 mg. The profiles of serum lipids such as total cholesterol (TC), high-density lipoprotein cholesterol (HDL) and triglyceride (TG) were observed. After 1 year administration TC had significantly decreased (p < 0.001) both in the TAM group and the TOR group, but no significant difference was found between these groups (p = 0.249). HDL had significantly decreased in the TAM group (p < 0.001), while it had significantly increased in the TOR group (p < 0.001), and a significant difference was found between the groups (p < 0.001). TG had significantly increased in the TAM group (p < 0.001) but significantly decreased in the TOR group (p < 0.001). The medication was switched in those who still had abnormal lipid metabolism and given to them for another year. After 1 year from the crossover TC and HDL had increased to the levels of before administration (p < 0.001) and TG had decreased in those (n = 57) whose medication was switched from TAM to TOR. While TC had decreased and TG had increased in those (n = 23) whose medication was switched from TOR to TAM (p < 0.001). The above findings have suggested that TOR provides better profiles of lipid metabolism than TAM.
10-05-2009, 10:34 AM
10-05-2009, 11:54 AM
10-05-2009, 12:09 PM
10-05-2009, 05:15 PM
10-05-2009, 05:43 PM
GOD, FAMILY, COUNTRY!!!
10-05-2009, 06:38 PM
ur theory is as good as any. The more I learn, the less I know LOL
....especially with the devil, oops I mean SD
10-06-2009, 03:25 AM
That would actually go for every strong non aromatising steroid.
What do you think about this: Strong non aromatising steroid shuts you down fast, no test to aromatise, no estrogen, possibly increases in aromatise enzyme, test comes back, estro aswell with a vengeance, combine with popular shbg lowering pct products and hello gyno.
10-06-2009, 09:45 AM
GOD, FAMILY, COUNTRY!!!
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