It should be Test/Tren. Both are potently lipolytic through divergent, as well as shared mechanisms (GR antagonism, prostaglindin activity changes, hormonally-mediated lipolysis, ridiculously-puissant nutrient repartitioning, inhibition of adipocyte differentiation via AR-interaction and genomic modification).
I don't know what anyone's beef with estrogen is on cuts either. Yeah, I recognize that it's not ideal aesthetically since water retention tends to skew how you actually perceive your rate of progress, but, all the same, local aromatization to estradiol in adipocyte tissue is a key mechanism through which testosterone exerts its lipolytic effects in subcutaneous fat.
To quote Bjontorp (or actually paraphrase because I don't have the study in front of me), who's done some really good research in the field:
"Test. & GH inhibit LPL and stimulate lipolysis markedly. Oestrogens seem to exert a net effect similar to Test."
And this is perfectly valid, because I've seen studies where testosterone administered in conjunction with an aromatase-inhibitor failed to reduce LPL as significantly as stand-alone testosterone.