Halo is good to end with. It is a major androgen that does not suppress at 30mg or less, so the theory holds in that case, however, it's exceptional. I've start many times with anadrol but would never end on it. In general, front with androgen and end with anabolics. Receptor binding must be minimal no matter what though at the end.
I would definetly have to disagree with that, in terms of suppression. Halo suppresses at a very small dose.
Even if it binds very weak at the AR, most these cause suppression through their metabolites. Case in point is Dbol. 17-methyl E2 is much more suppressive than diianabol itself. The same can be said with Halo and Drol (even the metbolite is not the same). We also have to look at receptor binding times on many of these. Even though it very weak binding to AR the binding time is not.
The only methylated steroid that could be used that is relatively weak in terms of suppression is Anavar but even that shows suppression at 2.5mg.
The effects of fluoxymesterone administration on testicular function.
Jones TM, Fang VS, Landau RL, Rosenfield RL.
Long term daily administration of fluoxymesterone (9alpha-fluoro-17alpha-methyl-11beta, 17beta-dihydroxyandrost-4-en-3-one) was associated with a modest suppression of sperm production and a profound suppression of testosterone levels in the absence of significant effects on plasma gonadotropin levels.
Nine normal male volunteers took either 10, 20, or 30 mg of fluoxymesterone daily for twelve weeks. Plasma samples were obtained for testosterone, estrogen, LH and FSH levels at biweekly intervals before, during and for up to 12 weeks after fluoxymesterone treatment. Samples were obtained for dehydroepiandrosterone sulfate, testosterone binding globulin and free testosterone assays at representative times before, during and after treatment. Although lower sperm counts were observed at several points during both the treatment and follow up periods, significant consistent suppression of spermatogenesis could not be demonstrated.
Reduced plasma testosterone levels were seen within 24 h after beginning fluoxymesterone, and further reductions were noted throughout the treatment period. Changes in plasma estrogen levels did not correlate with fluoxymesterone administration. Neither plasma LH nor plasma FSH levels were significantly altered by fluoxymesterone. A short term study utilizing a single dose of fluoxymesterone yielded similar findings. It is proposed that fluoxymesterone has a local effect on the Leydig cell which is not mediated by gonadotropins.
THis would only be applified in during peroids of conditional hypogonadism.
Not to be specific or anything. LOL I don't like this theory I think it's BS. Estrogen would be throug the roof which is not a great idea if you want your hpta to recover.
