[dujanw21]

i am looking for a good six month cycle and am wondering what other people think i should take with cjc-1295 (and how much cjc-1295 i should take). GOALS: I am looking to gain muscle, explosiveness, and height if possible. i am 20 and still think i can grow some since i have had some growing pains recently. i eat extremely healthy, drink sometimes but will stop. is cjc-1295 the right thing to take? what else should i take?

[papapumpsd]

Originally Posted by dujanw21

i am looking for a good six month cycle and am wondering what other people think i should take with cjc-1295 (and how much cjc-1295 i should take). GOALS: I am looking to gain muscle, explosiveness, and height if possible. i am 20 and still think i can grow some since i have had some growing pains recently. i eat extremely healthy, drink sometimes but will stop. is cjc-1295 the right thing to take? what else should i take?

A good 6-month cycle: Eat high protein, healthy fats, and complex carbs (higher GI carbs PWO), lift heavy, sleep. That's all you really need to know at 20.

[SoCo4Fun]

Mmmmmmmm....food.

[dujanw21]

i eat like that... im trying to get cut and big and most of all grow! even an inch i would be happy.

[papapumpsd]

Originally Posted by dujanw21

i eat like that... im trying to get cut and big and most of all grow! even an inch i would be happy.

Research 5x5 workouts then. At your age you should be able to put on some quality pounds w/the right food, workouts, and sleep. Do some research on AnabolicMinds on mass gaining and workouts, nutrition too. Lots of info around here.

[dujanw21]

when i say grow i mean grow in height... ya know? i can stil grow if i bumb my gh levels by a lot. what is the best cycle to do without side effects? 6 months of hgh with t3? or igf1 and cjc1295? or what? help me out?

[papapumpsd]

Originally Posted by dujanw21

when i say grow i mean grow in height... ya know? i can stil grow if i bumb my gh levels by a lot. what is the best cycle to do without side effects? 6 months of hgh with t3? or igf1 and cjc1295? or what? help me out?

I have NEVER seen a post regarding the use of GH or GH-releasers to literally grow vertically (height)! This is a bodybuilding forum and this section especially is devoted to bodybuilding and "anti-aging", not "how to get taller with GH".

What you're asking is sort of like me going to a forum for GH-deficient individuals who are using GH to improve the quality of life and asking, "How do I add a lot of muscle mass with GH? What should I use..hgh with t3? CJC/IGF-1? I'd be happy with 1 inch.....1 inch on my biceps...help me out?"

EDIT: I didn't mean for this to come across as rude or a$$holish. I do want to emphasize that while you can ask a slew of questions as a new member, that usually aggitates people because it makes you appear as though you are expecting us to spoonfeed you while you could have done your own research prior to posting and then asked more targeted, intelligent questions.

[Bobaslaw]

At 20, the chances are your Growth Plates are closed. Yes, there is the possibility that they are close to closed as some males can go up to age 25, but very rarely.
Best thing would be to get an x-ray of the wrist to determine if your plates are fully closed, or to what extent.
As far as GH (via HGH or via CJC/GHRPs) is concerned...
What you hope to achieve is the increase of growth factors for longitudinal bone growth. Growth factors including but not limited to Osteocalcin, which increases osteoblast activity, as well as IGF-1.
There are optimum protocols for GHD in children with SS (Short Stature), none of them which yet encompass CJC or GHRP. So far, HGH, GHRH and rhIGF have been used in treatment.
I surmise CJC/GHRP could produce a beneficial increase in Growth Factors for this purpose, however, it is just speculation on what protocol and dosages would be best.
I personally believe that pulsitility is key to this topic and that GHRH and GHRP may be a good combination.
I have no idea about the elevated troughs in CJC or whether that would be a detriment for optimal signaling pathway resensitization especially for optimal osteocalcin.
Anyhow this is just info in general. I can't say what is best.

Figure out if your GP are closed before doing anything in this direction...
Do some research into GHD and Short Stature.

Take Care.

[datBtrue]

Originally Posted by dujanw21

...and height if possible. i am 20 and still think i can grow some since i have had some growing pains recently.

Why all these questions from kids that want to grow?

Why do they ALL think growth hormone is the answer?

Because they are all incapable of doing the least bit of research to try to figure it out. There is no easy text book answer. I know that if you put the word "growth" in something the masses think that something will make you grow. But nothing takes place in a vaccum.

From, The Roles of Oestrogen in the Male by Richard M. Sharpe.



I'll give you a hint. The intra-cellular signalling is important.

"Conversely, 1, 25 dihydroxy vitamin D3 prolongs GH signalling in osteoblast-like cells by inhibition of SOCS-3 and CIS expression which might contribute to bone turnover."

Go read "Seeking SOCS and sex steroids", Michael J. Waters.

Here is the opening paragraph:

In seeking to explain why oral estrogen inhibits the GH–IGF-I axis, a recent study has unearthed a new way that steroid hormones can influence growth hormone action. This involves suppressors of cytokine signalling (SOCS), which offer a new level of understanding in signal control and crosstalk.

[datBtrue]

Originally Posted by Bobaslaw

...There are optimum protocols for GHD in children with SS (Short Stature), none of them which yet encompass CJC or GHRP. So far, HGH, GHRH and rhIGF have been used in treatment....

Yes but the KEY is there is already a LOT going on in the body of a child. For a child often GH is what is needed because they have a mutation that precludes them from producing their own but the other factors necessary for growth will occur as they do with any child.

The problem becomes much, much, much more complex when a 20 year old needs to duplicate all that is happening in a child poised for growth. That can not be done ...or it can but it takes changing their "young adult" sex hormone profile and duplicating the pattern of a child in adolescence. Not easy and it may not even be desirable.

The same thing happens when an adult realizes DHT transdermal is used to treat micropenis in children. They think they can use it to become larger. But they can not because those receptors responsible for growth are not responsive much beyond 14 - 15 years of age. Instead the DHT receptors in that region in an adult who uses such a locally applied transdermal uptakes DHT and enlarges the prostate. But that doesn't mean idiots don't try such things.

[xtraflossy]

Originally Posted by datBtrue

Yes but the KEY is there is already a LOT going on in the body of a child. For a child often GH is what is needed because they have a mutation that precludes them from producing their own but the other factors necessary for growth will occur as they do with any child.

The problem becomes much, much, much more complex when a 20 year old needs to duplicate all that is happening in a child poised for growth. That can not be done ...or it can but it takes changing their "young adult" sex hormone profile and duplicating the pattern of a child in adolescence. Not easy and it may not even be desirable.

The same thing happens when an adult realizes DHT transdermal is used to treat micropenis in children. They think they can use it to become larger. But they can not because those receptors responsible for growth are not responsive much beyond 14 - 15 years of age. Instead the DHT receptors in that region in an adult who uses such a locally applied transdermal uptakes DHT and enlarges the prostate. But that doesn't mean idiots don't try such things.

Well there go all the kids' dream right there, in one visit.




I must spread the love before....

[SoCo4Fun]

Holy crap...there is something called micropenis? That sounds like a name I would call my brother to get him to stop being a pussy and lift more weight.

[Bobaslaw]

Originally Posted by datBtrue

Yes but the KEY is there is already a LOT going on in the body of a child. For a child often GH is what is needed because they have a mutation that precludes them from producing their own but the other factors necessary for growth will occur as they do with any child.

The problem becomes much, much, much more complex when a 20 year old needs to duplicate all that is happening in a child poised for growth. That can not be done ...or it can but it takes changing their "young adult" sex hormone profile and duplicating the pattern of a child in adolescence. Not easy and it may not even be desirable.

The same thing happens when an adult realizes DHT transdermal is used to treat micropenis in children. They think they can use it to become larger. But they can not because those receptors responsible for growth are not responsive much beyond 14 - 15 years of age. Instead the DHT receptors in that region in an adult who uses such a locally applied transdermal uptakes DHT and enlarges the prostate. But that doesn't mean idiots don't try such things.

Totally agree, Dat.

Most of what I was conveying was with direct regards to Growth Hormone Deficiency (GHD) in children with Short Stature so it may come across misleading when viewed by people that may not fit that "mold".
I am glad you are bringing up this distinction so that people can be aware of the bigger picture and the other factors at play. Especially, people that think GH is the single answer to height growth inspite of the fact they may not actually have GHD.

Instead the DHT receptors in that region in an adult who uses such a locally applied transdermal uptakes DHT and enlarges the prostate. But that doesn't mean idiots don't try such things.

All the people with a micropenis AND a grapfruit sized prostate raise your hands, lol

[datBtrue]

Originally Posted by SoCo4Fun

Holy crap...there is something called micropenis? That sounds like a name I would call my brother to get him to stop being a pussy and lift more weight.

When I was in law school there was a guy I knew who got drunk one night and was really depressed. He didn't know what to do about this girl who had the hots for him...

...take her home and have a little hobble-dah-gee...make her your girlfriend...whatever...that was the simple Dat solution.

"No Dat you don't understand I have a small penis" he said. His roomate looked at me like "yes he does."

So Dat being Dat said "hey bro don't worry about it...size doesn't matter...you know there is a range of normal..."

"No Dat I am really small" he mumbles. So I say "Hey c'mon don't be depressed, you know most of the nerves are located in the first few inches of a women's..."

"NO Dat, you don't understand! My erection is 1 inch!"

His roomy looks at me and shakes his head and says "...yep...maybe smaller...he has a tiny mirco penis."

Silence...for the only time in my life I had absolutely nothing to say. I mean WOW! What do you do with THAT!

This guy had everything in the world going for him. He was smart, he was going to make plenty of money, he was handsome & had a fairly muscular body (at the time I even wished that I was that muscular)... plus he was one of the nicest guys I've ever met in my life.

But he had a micropenis...and nothing in the world was ever going to change that fact...NOTHING!

It kind of makes fantasy aspirations to become "John Holmes" seem stupid when you find out there is someone in the world that just wants an actual functional erection...a small normal sized penis is his desperate aspiration.

[datBtrue]

Catch-Up Growth after Glucocorticoid Excess: A Mechanism Intrinsic to the Growth Plate, J Baron, KO Klein, MJ Colli, JA Yanovski, JA Novosad, JD Bacher and GB Cutler Jr , Endocrinology, 1994 Vol 135, 1367-1371

In humans and other mammals, the release from growth-inhibiting conditions, such as glucocorticoid excess, leads to supranormal linear growth. The prevailing explanation for this catch-up growth involves a central nervous system mechanism that compares actual body size to an age-appropriate set-point and adjusts growth rate accordingly via a circulating factor.

Although such a neuroendocrine "sizostat" was hypothesized more than 30 yr ago, its existence has never been confirmed experimentally. Here we show that suppression of growth within a single growth plate by locally administered glucocorticoid is followed by local catch-up growth that is restricted to the affected growth plate. Thus, the catch-up growth cannot be explained by neuroendocrine mechanism but, rather, must arise from a mechanism intrinsic to the growth plate.

To explain this finding, we propose that the normal senescent decline in growth plate function depends not on age per se, but on the cumulative number of stem cell divisions, and that glucocorticoid administration, by suppressing stem cell proliferatio