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    Slintensity Feedback, Reviews, Questions


    Slintensity is out and we think it is the most effective GDA on the market. We want your questions, your experiences, your reviews of our exciting new product. We worked hard to get this to the market with the most potent ingredients and not break your bank account. GDA's are different than other products we have released so far and the use of them can be much more varied than most other product categories.

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    Matt hits the books to come up with gold:

    J Microbiol Biotechnol. 2012 Jan;22(1):147-55.

    Synergic effects of bitter melon and β-Glucan composition on STZ-induced rat diabetes and its complications.

    Kim JW, Cho HR, Moon SB, Kim KY, Ku S.
    Source

    Glucan Corp. Research Institute, Marine Biotechnology Center, Busan 617-763, Korea.

    Abstract

    β-Glucan purified from oats (OG) and bitter melon, Momordica charantia Linn (MC), water extracts have shown favorable effects on diabetes and its complications. We investigated to find out the optimal composition showing hypoglycemic and antidiabetic complication effects in variable compositions (OG:MC = 1:1, 1:2, 1:4, 1:6, 1:8, 1:10, 2:1, 4:1, 6:1, 8:1, 10:1). Extracts were administered orally once a day for 28 days following 7 days post streptozotocin (STZ) dosing. Five rats per group (total 15 groups; Intact, STZ, OG, MC, and the variable composition groups) were selected according to the blood glucose and body weight at 6 days after STZ dosing. After 28 days of extracts dosing, the changes on the body weight, liver and kidney weight, blood glucose, blood urea nitrogen (BUN), creatinine, aspartate aminotransferase (AST) and alanine aminotransferase (ALT), low-density lipoprotein (LDL), and total-cholesterol levels were observed. As the result of STZ-induced diabetes, decreases of body weight, increases of the liver and kidney weights, blood glucose, BUN, creatinine, AST, ALT, LDL, and total-cholesterol levels in STZ control were detected compared with intact control. However, these changes of hyperglycemia, diabetic nephropathy, hepatopathy, and hyperlipemia were dramatically decreased in the OG and MC single-dosing group, and all composition groups. In addition, there were more favorable effects in all composition groups compared with the OG and MC single-dosing groups. Among variable compositions, the OG:MC 1:2 mixed group showed the most synergic effects in this study.

    PMID:22297232 [PubMed - indexed for MEDLINE]
    Free full text


    Bitter melon extracts in diabetic and normal rats favorably influence blood glucose and blood pressure regulation.

    Clouatre DL, Rao SN, Preuss HG.
    Source

    Glykon Technologies Group, LLC, Santa Monica, California, USA. dallasclouatre@mac.com

    Abstract

    Bitter melon (BM) was tested in normal and streptozotocin (STZ)-induced diabetic rats. First, normal and diabetic Wistar rats were given four test extracts (EX-1-EX-4) of a wild-genotype BM or metformin by intubation. Second, normal Sprague-Dawley rats were divided into control and three test groups given for 52 days one of three BM preparations in food: Chinese or Indian commercial preparations or EX-4 from experiment I. In experiment I, extracts of BM administered at 50 mg/kg of body weight in normal rats reduced blood sugar for 4 hours without, unlike metformin, inducing hypoglycemia. In STZ-induced diabetic rats, two extracts administered at 250 mg/kg decreased glucose levels to values comparable to metformin at 150 mg/kg. At 4 hours, EX-1 and EX-4 significantly reduced blood glucose 67% and 63%, respectively, compared with metformin's 54%. In experiment II, all test groups had lowered systolic, but not diastolic, blood pressure. The China and EX-4 arms had significantly lowered serum glucose levels compared with the control. In the glucose tolerance test, only EX-4 had significantly lowered glucose levels. Only EX-4 had significantly lowered angiotensin converting enzyme (ACE) activity. All active arms showed significance in the losartan challenge (the renin-angiotensin system [RAS]), with the greatest effect in the EX-4 group. In the N(ω)-nitro-l-arginine-methyl ester challenge, only EX-4 exhibited a significant impact on the nitric oxide system, suggesting higher activity in this group. In the STZ-induced diabetic rat model, wild-type BM powerfully lowered glucose levels, and, in healthy adult rats, wild-type BM exhibited beneficial effects in the regulation of blood glucose, in RAS and ACE inhibition, and in nitric oxide generation.

    The combination of our special extract and the Norvaline will have a potent effect on NO generation and vascular dilation.
    J Nutr Biochem. 2011 Nov;22(11):1064-73. Epub 2011 Jan 28.
    Bioactives from bitter melon enhance insulin signaling and modulate acyl carnitine content in skeletal muscle in high-fat diet-fed mice.

    Wang ZQ, Zhang XH, Yu Y, Poulev A, Ribnicky D, Floyd ZE, Cefalu WT.
    Source

    Center for the Study of Botanicals and Metabolic Syndrome, Pennington Biomedical Research Center, LSU System, Baton Rouge, LA 70808, USA. wangzq@PBRC.edu

    Abstract

    Bioactive components from bitter melon (BM) have been reported to improve glucose metabolism in vivo, but definitive studies on efficacy and mechanism of action are lacking. We sought to investigate the effects of BM bioactives on body weight, muscle lipid content and insulin signaling in mice fed a high-fat diet and on insulin signaling in L6 myotubes. Male C57BL/6J mice were randomly divided into low-fat diet control (LFD), high-fat diet (HFD) and HFD plus BM (BM) groups. Body weight, body composition, plasma glucose, leptin, insulin and muscle lipid profile were determined over 12 weeks. Insulin signaling was determined in the mouse muscle taken at end of study and in L6 myotubes exposed to the extract. Body weight, plasma glucose, insulin, leptin levels and HOMA-IR values were significantly lower in the BM-fed HFD group when compared to the HFD group. BM supplementation significantly increased IRS-2, IR β, PI 3K and GLUT4 protein abundance in skeletal muscle, as well as phosphorylation of IRS-1, Akt1 and Akt2 when compared with HFD (P<.05 and P<.01). BM also significantly reduced muscle lipid content in the HFD mice. BM extract greatly increased glucose uptake and enhanced insulin signaling in L6 myotubes. This study shows that BM bioactives reduced body weight, improved glucose metabolism and enhanced skeletal muscle insulin signaling. A contributing mechanism to the enhanced insulin signaling may be associated with the reduction in skeletal muscle lipid content. Nutritional supplementation with this extract, if validated for human studies, may offer an adjunctive therapy for diabetes.
    Copyright © 2011 Elsevier Inc. All rights reserved.

    Eur J Nutr. 2012 May 19. [Epub ahead of print]

    4-Hydroxyisoleucine stimulates glucose uptake by increasing surface GLUT4 level in skeletal muscle cells via phosphatidylinositol-3-kinase-dependent pathway.

    Jaiswal N, Maurya CK, Venkateswarlu K, Sukanya P, Srivastava AK, Narender T, Tamrakar AK.
    Source

    Division of Biochemistry, CSIR-Central Drug Research Institute, Lucknow, 226001, India.

    Abstract

    PURPOSE:

    To determine the effect of 4-Hydroxyisoleucine (4-HIL), an unusual amino acid isolated from the seeds of Trigonella foenum-graecum, on glucose uptake and the translocation of glucose transporter 4 (GLUT4) to plasma membrane in skeletal muscle cells and to investigate the underlying mechanisms of action.
    METHODS:

    Rat skeletal muscle cells (L6-GLUT4myc) were treated with 4-HIL, and the effect on glucose uptake was determined by measuring the incorporation of radio-labeled 2-deoxy-[(3)H]-D: -glucose (2-DG) into the cell. Translocation of GLUT4myc to plasma membrane was measured by an antibody-coupled colorimetric assay.
    RESULTS:

    The prolonged exposure (16 h) of L6-GLUT4myc myotubes to 4-HIL caused a substantial increase in the 2-DG uptake and GLUT4 translocation to the cell surface, without changing the total amount of GLUT4 and GLUT1. Cycloheximide treatment reversed the effect of 4-HIL on GLUT4 translocation to the basal level suggesting the requirement of new protein synthesis. The 4-HIL-induced increase in GLUT4 translocation was completely abolished by wortmannin, and 4-HIL significantly increased the basal phosphorylation of AKT (Ser-473), but did not change the mRNA expression of AKT, IRS-1, GLUT4, and GSK3β.
    CONCLUSION:

    Results suggest that 4-HIL stimulates glucose uptake in L6-GLUT4myc myotubes by enhancing translocation of GLUT4 to the cell surface in a PI-3-kinase/AKT-dependent mechanism.

    PMID:22610671 [PubMed - as supplied by publisher]

    Adipocyte-Derived Th2 Cytokines and Myeloid PPARδ Regulate Macrophage Polarization and Insulin Sensitivity

    Kihwa Kang1, Shannon M. Reilly1, Volkan Karabacak1, Matthew R. Gangl1, Kelly Fitzgerald1, Ben Hatano1, 2 and Chih-Hao Lee1, ,
    1 Department of Genetics and Complex Diseases, Division of Biological Sciences, Harvard School of Public Health, Boston, MA 02115, USA

    Corresponding author

    2 Present address: Department of Pathology, Japan Self-Defense Forces Central Hospital, 1-2-24 Ikejiri, Tokyo 154-0001, Japan




    Summary

    The polarization of adipose tissue-resident macrophages toward the alternatively activated, anti-inflammatory M2 phenotype is believed to improve insulin sensitivity. However, the mechanisms controlling tissue macrophage activation remain unclear. Here we show that adipocytes are a source of Th2 cytokines, including IL-13 and to a lesser extent IL-4, which induce macrophage PPARδ/β (Ppard/b) expression through a STAT6 binding site on its promoter to activate alternative activation. Coculture studies indicate that Ppard ablation renders macrophages incapable of transition to the M2 phenotype, which in turns causes inflammation and metabolic derangement in adipocytes. Remarkably, a similar regulatory mechanism by hepatocyte-derived Th2 cytokines and macrophage PPARδ is found to control hepatic lipid metabolism. The physiological relevance of this paracrine pathway is demonstrated in myeloid-specific PPARδ−/− mice, which develop insulin resistance and show increased adipocyte lipolysis and severe hepatosteatosis. These findings provide a molecular basis to modulate tissue-resident macrophage activation and insulin sensitivity.

    The addition of fenugreek extract (Trigonella foenum-graecum) to glucose feeding increases muscle glycogen resynthesis after exercise.

    Ruby BC, Gaskill SE, Slivka D, Harger SG.
    Source

    Department of Health and Human Performance, The University of Montana, Missoula, Montana 59812-1825, USA. brent.ruby@mso.umt.edu

    Abstract

    The purpose of this study was to determine the effects of ingesting an oral supplement containing 4-Hydroxyisoleucine (4-OH-Ile, isolated from fenugreek seeds [Trigonella foenum-graecum]) with a glucose beverage on rates of post-exercise muscle glycogen resynthesis in trained male cyclists. Following an overnight fast (12 hr), subjects completed a 90-minute glycogen depletion ride after which a muscle biopsy was obtained from the vastus lateralis. Immediately and 2 hours after the muscle biopsy, subjects ingested either an oral dose of dextrose (Glu) (1.8 g.kg BW(-1)) or 4-OH-Ile supplement (Glu+4-OH-Ile, including 2.0 mg.kg(-1) 4-OH-Ile with the same oral dose of dextrose) with a second muscle biopsy 4 hours after exercise. Post exercise muscle glycogen concentration was similar for both trials. Overall, there was a significant increase in glucose and insulin concentrations from time 0 throughout the majority of the 4-hour recovery period, with no significant differences between the two trials at any time point. Although muscle glycogen concentration significantly increased from immediately post exercise to 4 hr of recovery for both trials, the net rate of muscle glycogen resynthesis was 63% greater during Glu+4-OH-Ile (10.6+/-3.3 vs. 6.5+/-2.6 g.kg wet wt.(-1).hr.(-1) for the Glu+4-OH-Ile and Glu trials, respectively). These data demonstrate that when the fenugreek extract supplement (4-OH-Ile) is added to a high oral dose of dextrose, rates of post-exercise glycogen resynthesis are enhanced above dextrose alone.

    PMID:15719265 [PubMed - indexed for MEDLINE]


    Activating effect of momordin, extract of bitter melon (Momordica Charantia L.), on the promoter of human PPARdelta.

    Sasa M, Inoue I, Shinoda Y, Takahashi S, Seo M, Komoda T, Awata T, Katayama S.
    Source

    Department of Diabetes and Endocrinology, Saitama Medical University, Japan.

    Abstract

    AIM:

    Bitter melon (Momordica charantia L.) is a common vegetable grown in Okinawa that has also been used recently in medicine for the treatment of diseases such as diabetes, hypertension, and dyslipidemia. Among Bitter melon extracts compounds, we focused on an extract known as momordin in the present study, to examine its effect on peroxisome-proliferator activated-receptor (PPAR) delta (also called PPARdelta in rodents) expression and promoter activity of the human PPARdelta gene.
    METHODS:

    A human PPARdelta promoter-reporter plasmid was made as a template from a BAC CLONE (RPCI-11C) containing a -3076 bp (BglI site) +74 bp (EcoRI site) sequence. Luciferase assay of PPARdelta promoter activity was performed using HepG2 cells.
    RESULTS:

    10 and 25 nM Momordin significantly increased the expression of PPARdelta mRNA 1.5-fold (relative to the control). Moreover, 10 and 25 nM Momordin significantly increased PPARdelta promoter activity in a dose-dependent manner, reaching more than 1.5-fold relative to the control.
    CONCLUSION:

    Our present data obtained through successful cloning of the PPARdelta promoter demonstrate that PPARdelta production and activation are upregulated through PPARdelta promoter activity following momordin treatment.

    PMID:20032574 [PubMed - indexed for MEDLINE]
    Tannic acid stimulates glucose transport and inhibits adipocyte differentiation in 3T3-L1 cells.

    Liu X, Kim JK, Li Y, Li J, Liu F, Chen X.
    Source

    Department of Biochemistry, Edison Biotechnology Institute, College of Osteopathic Medicine, Ohio University, Athens, OH 45701, USA.

    Abstract

    Obesity is a major risk factor for Syndrome X and type II diabetes (T2D). However, most antidiabetic drugs that are hypoglycemic also promote weight gain, thus alleviating one symptom of T2D while aggravating a major risk factor that leads to T2D. Adipogenesis, the differentiation and proliferation of adipocytes, is a major mechanism leading to weight gain and obesity. It is highly desirable to develop pharmaceuticals and treatments for T2D that reduce blood glucose levels without inducing adipogenesis in patients. Previously, we reported that an extract from Lagerstroemia speciosa L. (banaba) possessed activities that both stimulated glucose transport and inhibited adipocyte differentiation in 3T3-L1 cells. Using glucose uptake assays and Western/Northern blot analyses as major tools and 3T3-L1 cells as a model, we showed that the banaba extract (BE) with tannin removed was devoid of the 2 activities, and tannic acid (TA), a major component of tannins, had the same 2 activities as BE. Inhibitors known to abolish insulin-induced glucose transport also blocked TA-induced glucose transport. We further detected that TA induced phosphorylation of the insulin receptor (IR) and Akt, as well as translocation of glucose transporter 4 (GLUT 4), the protein factors involved in the signaling pathway of insulin-mediated glucose transport. We also demonstrated that TA inhibited the expression of key genes for adipogenesis. Differences between samples with or without TA in all of the quantitative assays were significant (P < 0.05). These results suggest that TA may be useful for the prevention and treatment of T2D and its associated obesity. TA may have the potential to become the lead compound in the development of new types of antidiabetic pharmaceuticals that are able to reduce blood glucose levels without increasing adiposity
    and this...


    Antidiabetes and Anti-obesity Activity of Lagerstroemia speciosa.

    Klein G, Kim J, Himmeldirk K, Cao Y, Chen X.
    Source

    College of Osteopathic Medicine, Edison Biotechnology Institute, Department of Chemistry and Biochemistry, The Molecular and Cellular Biology Program, Department of Biological Science and Department of Biomedical Science, Ohio University, USA.

    Abstract

    The leaves of Lagerstroemia speciosa (Lythraceae), a Southeast Asian tree more commonly known as banaba, have been traditionally consumed in various forms by Philippinos for treatment of diabetes and kidney related diseases. In the 1990s, the popularity of this herbal medicine began to attract the attention of scientists worldwide. Since then, researchers have conducted numerous in vitro and in vivo studies that consistently confirmed the antidiabetic activity of banaba. Scientists have identified different components of banaba to be responsible for its activity. Using tumor cells as a cell model, corosolic acid was isolated from the methanol extract of banaba and shown to be an active compound. More recently, a different cell model and the focus on the water soluble fraction of the extract led to the discovery of other compounds. The ellagitannin Lagerstroemin was identified as an effective component of the banaba extract responsible for the activity. In a different approach, using 3T3-L1 adipocytes as a cell model and a glucose uptake assay as the functional screening method, Chen et al. showed that the banaba water extract exhibited an insulin-like glucose transport inducing activity. Coupling HPLC fractionation with a glucose uptake assay, gallotannins were identified in the banaba extract as components responsible for the activity, not corosolic acid. Penta-O-galloyl-glucopyranose (PGG) was identified as the most potent gallotannin. A comparison of published data with results obtained for PGG indicates that PGG has a significantly higher glucose transport stimulatory activity than Lagerstroemin. Chen et al. have also shown that PGG exhibits anti-adipogenic properties in addition to stimulating the glucose uptake in adipocytes. The combination of glucose uptake and anti-adipogenesis activity is not found in the current insulin mimetic drugs and may indicate a great therapeutic potential of PGG


    It's not all about the Corosolic Acid.
    Get your GlycoMyx on!

    What would be a good source of carbs to take with Slintensity, you ask? What about Zoidberg?

    Zhejiang Da Xue Xue Bao Yi Xue Ban. 2011 Jul;40(4):374-9.
    [Effect of purple sweet potato flavonoids on metabolism of glucose and lipids in diabetic rats].

    [Article in Chinese]
    Jiang HF, Li XR, Tang C.
    Source

    School of Medicine and Life Sciences, Zhejiang University City College, Hangzhou 310015, China.

    Abstract

    OBJECTIVE:

    To investigate the effects of purple sweet potato flavonoids (PSPF) on blood glucose and lipids levels in diabetic rats.
    METHODS:

    Diabetes was induced by intraperitoneal injection of streptozotocin (STZ, 65 mg.kg(-1)) in rats. The changes of fasting blood glucose and lipids levels in serum and body weight, food and fluid intake of diabetic rats treated with PSPF were examined.
    RESULTS:

    Diabetic symptoms were ameliorated after rats were fed with PSPF. The fasting blood glucose (FBG), GSP, TC, TG, LDL-C were decreased and serum HDL-C levels were increased (P<0.01) in high, medium dose PSPF groups; while FBG, serum GSP, TG, LDL-C were also improved in low dose group (P<0.05 or P<0.01).
    CONCLUSION:

    Purple sweet potato flavonoids can decrease the blood glucose and lipids levels in diabetic rats.

    PMID:21845749 [PubMed - indexed for MEDLINE]
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    Quote Originally Posted by Geoforce View Post
    Slintensity is out and we think it is the most effective GDA on the market. We want your questions, your experiences, your reviews of our exciting new product. We worked hard to get this to the market with the most potent ingredients and not break your bank account. GDA's are different than other products we have released so far and the use of them can be much more varied than most other product categories.

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    Give me a bottle so I don't break my bank account and I will give you some feedback, a review, and some questions. Deal?
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    Word. If I get a bottle ill def give a review
    [/COLOR]I'm a Brooklyn boy I may take some gettin' use to
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    Quote Originally Posted by T-Bone
    <img src="http://anabolicminds.com/forum/attachment.php?attachmentid=67 245"/>
    Me too
    [/COLOR]I'm a Brooklyn boy I may take some gettin' use to
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    Free loaders
    Serious Nutrition Solutions Representative
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    Quote Originally Posted by halfhuman View Post
    Me too

    You're only half human though and this product is more for humans. Plus Dad likes me more.
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    Quote Originally Posted by Distilled Water View Post
    Free loaders

    Yeah, take a look at my profile!.
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    I'll purchase some though eventually.
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    I'm bumping this thread. Free publicity!
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    Slintastic! Slinabate


    Slintensity- Force feed your muscles for maximum swole-ness.
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    I'm sure we will run sponsored Slintensity logs, I just don't know how soon it will be. I don't get to make those kind of decisions. I pick out the music for the car rides.
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    Quote Originally Posted by Geoforce
    I'm sure we will run sponsored Slintensity logs, I just don't know how soon it will be. I don't get to make those kind of decisions. I pick out the music for the car rides.
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    Looks like initial feedback of Slintensity is pretty positive:

    Just took my first dose of slintensity before my Pre workout meal. 60 grams carbs from oats, 10 egg whites.

    This stuff rocks! The pump was insane. I didn't feel bloated at all during my workout! My stomach felt empty which is rare when I eat carbs. Iv tried glycobol and slin sane, slintensity is by far my favorite gda
    ~5hrs after first dose of slintensity and glycomyx (downed some in between work and classes) - fullness, vascularity definitely noticeable.
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    Quote Originally Posted by Geoforce View Post
    I pick out the music for the car rides.

    lucky sob!
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    Quote Originally Posted by criticalbench View Post
    lucky sob!
    Matt and Blake love some Michael Bolton.
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    Quote Originally Posted by Geoforce View Post
    I'm sure we will run sponsored Slintensity logs, I just don't know how soon it will be. I don't get to make those kind of decisions. I pick out the music for the car rides.
    I have no doubt we'll have some logs in the future, but in the meantime, how can you pass up the current deal:

    http://www.nutraplanet.com/product/s...-glycomyx.html

    Slintensity + GlycoMyx for under $30 is an incredible steal, and hardly breaks the bank account; especially when you know the strength of Slintensity and being able to dose 60g carbs with just 1 cap.

    And then GlycoMyx turns any protein shake into an MRP if you want it to. And if mixed with the right chocolate protein, it tastes like Coco Wheats lol (srs).
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    Quote Originally Posted by Geoforce View Post
    Matt and Blake love some Michael Bolton.
    I'm a bigger Michael McDonald fan, actually.

    (he's a bigger Michael McDonald fan, actuallllllyyyyy!)
    Psalm 34:10 - "The lions may grow weak and hungry, but those who seek the Lord lack no good thing."
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    I got a fever and the only prescription is more feedback!

    (and cowbell)
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    Ok, picked up a couple bottles from the NP deal and have a few questions. What's the half life purr cap? And how critical is the 60g carb threshold? Many thanks!
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    Quote Originally Posted by lava135 View Post
    Ok, picked up a couple bottles from the NP deal and have a few questions. What's the half life purr cap? And how critical is the 60g carb threshold? Many thanks!
    Not sure about what you're asking with half-life, but you'll want to eat within 15-20 minutes of dosing. And the 60g limit is something you'll have to keep an eye on. I know Matt has been experimenting a bit, and it's very easy to go hypo if you don't get the carbs in. For safety reasons we recommend 60g.

    Let us know what you think of it!
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    60 grams kinda covers our butt. I'm certainly going to experiment with a bit lower perhaps (I don't think I'd die from say 50 grams) but it isn't something we are actually recommending if you catch my drift.
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    Quote Originally Posted by MidwestBeast

    Not sure about what you're asking with half-life, but you'll want to eat within 15-20 minutes of dosing. And the 60g limit is something you'll have to keep an eye on. I know Matt has been experimenting a bit, and it's very easy to go hypo if you don't get the carbs in. For safety reasons we recommend 60g.

    Let us know what you think of it!
    well if i take a cap post w my shake and then have breakfast an hour or so later, should i pop another?
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    Quote Originally Posted by Geoforce
    60 grams kinda covers our butt. I'm certainly going to experiment with a bit lower perhaps (I don't think I'd die from say 50 grams) but it isn't something we are actually recommending if you catch my drift.
    got it -this probably answers my half life question as well
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    Quote Originally Posted by lava135 View Post
    well if i take a cap post w my shake and then have breakfast an hour or so later, should i pop another?
    I would make dosing be dependent on carb intake. I wouldn't think with this scenario you'd need to pop another. People can feel free to use different protocols and do their own experimenting and see what works. Personally I'm not going to use mine immediately post-workout (insulin sensitivity already elevated from exercise), I'm going to use it mainly before big carb meals to keep blood sugar stable and occasionally pre-workout with GlycoMyx.
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    Quote Originally Posted by Geoforce View Post
    Personally I'm not going to use mine immediately post-workout (insulin sensitivity already elevated from exercise), I'm going to use it mainly before big carb meals to keep blood sugar stable and occasionally pre-workout with GlycoMyx.
    Bingo.

    Same here.
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    I'm curious on insulin spike post workout. How long after do you wait for post meal? What's the time frame? For example after workout u have your protein shake than for some reason u don't eat for another 1,2,3 hours? Is your insulin still spiked for that long? I would think no more than 90 min it would be spiked. Anyone knows the time frame?
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    Quote Originally Posted by halfhuman View Post
    I'm curious on insulin spike post workout. How long after do you wait for post meal? What's the time frame? For example after workout u have your protein shake than for some reason u don't eat for another 1,2,3 hours? Is your insulin still spiked for that long? I would think no more than 90 min it would be spiked. Anyone knows the time frame?
    The debate has always been on the "anabolic window." IIRC, they say it's around 90 minutes post-workout. Me, personally, I like to slam my fast-digesting carbs and protein almost right after workout (immediately after longer workouts or maybe 15 minutes after ones that aren't as long). And then about an hour and a half or 2 hours, later, I eat a whole food meal with slow digesting carbs and protein and some good fats in there, too.

    That's just me.
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    Would it be safe to take 1 cap for each meal, taking in 4 meals a day as long as carb intake is 60g on each meal?
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    Quote Originally Posted by python93 View Post
    Would it be safe to take 1 cap for each meal, taking in 4 meals a day as long as carb intake is 60g on each meal?
    Safe? Yes. Though unless you are doing some major cardio it would probably be a waste.
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    Quote Originally Posted by dsade

    Safe? Yes. Though unless you are doing some major cardio it would probably be a waste.
    Are you saying that from a perspective of cutting is what the product is intended for, or that there would be no point in taking it in a caloric surplus?
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    Quote Originally Posted by uvawahoowa View Post
    Are you saying that from a perspective of cutting is what the product is intended for, or that there would be no point in taking it in a caloric surplus?
    No, I am saying with a product designed to increase uptake at muscle (among other things), that your muscle can only pick up so much per day unless you are doing major depletion workouts.
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    Anything beneficial to stacking SlinTensity and SSV2 together with GlycoMyx, preworkout? Or would that just be overkill and/or unnecessary?

    Or what if I added some bulk Na-R-Ala to SlinTensity?
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    Quote Originally Posted by dsade

    No, I am saying with a product designed to increase uptake at muscle (among other things), that your muscle can only pick up so much per day unless you are doing major depletion workouts.
    Would you still benefit from faster glucose clearing and lowered insulogenic response with that many caps/day? Or are you saying that there is just a diminished benefit past say 2-3 caps/day?
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    Quote Originally Posted by uvawahoowa View Post
    Would you still benefit from faster glucose clearing and lowered insulogenic response with that many caps/day? Or are you saying that there is just a diminished benefit past say 2-3 caps/day?
    Yes, you would get that if you were on a pretty dramatic cut.
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    Quote Originally Posted by BigGame84 View Post
    Anything beneficial to stacking SlinTensity and SSV2 together with GlycoMyx, preworkout? Or would that just be overkill and/or unnecessary?

    Or what if I added some bulk Na-R-Ala to SlinTensity?
    You can definitely add in bulk na-R-ALA.
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    Quote Originally Posted by dsade View Post
    You can definitely add in bulk na-R-ALA.

    Would you recommend bulk na-R-ALA or NP's Yellow Gold to stack it with?
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    Quote Originally Posted by BigGame84 View Post
    Would you recommend bulk na-R-ALA or NP's Yellow Gold to stack it with?
    I worked hard to make sure there were no GI problems with Slintensity...not sure I would recommend the YG. Just pick up a reliable, and cheap, Na-R-ALA.
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    Quote Originally Posted by dsade View Post
    I worked hard to make sure there were no GI problems with Slintensity...not sure I would recommend the YG. Just pick up a reliable, and cheap, Na-R-ALA.
    Looks like NP has some bulk Na-R-ALA in stock. I'll probably go that direction. Thanks.
  

  
 

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