really, product of the year? interesting....what are the major benefits of formestane? do you find it puts you in a good mood?
Make sure to open the bottle up and pour out any the pump doesnt reach. The straw only reaches so far and the bottom is domed so...
the straw wont get everything.
Formestane reduces estrogen which in turn elevates testosterone levels.
Formestane is also anabolic unlike any other Aromatase inhibitor.
Great for on cycle, PCT, or stand alone.
If your testosterone levels are low it will certainly effect mood.
so it sounds like it could be productive for a semi natty stack with 4-dhea- a good one though of course
Formeron, Prolactrone, DAA is an amazing stack.
Prolactrone significantly increases Growth hormone levels.
In this study you will see that Ldopa administered concurrent with a decarboxylase inhibitor increases growth hormone levels.
The level of increase seen in this study was comparable to 5-7 IU injected growth hormone.
Eur J Pediatr. 1976 Jun 8;122(3):195-200.
[HGH secretion after oral application of L-dopa and L-carbidopa (author's transl)].
[Article in German]
Sch?nberger W, Ziegler R, Brodt B, Grimm W.
The stimulatory effect of L-Dopa and L-Carbidopa (Nacom) on HGH secretion was determined in 12 children of normal height aged from 6 to 14 years. Each child received a standard dose of 250 mg L-Dopa and 25 mg L-Carbidopa p.o. HGH concentration in the serum was determined at standard intervals. All subjects showed a sufficient increase of HGH. The mean value was 19.6 ng/ml. According to the maximum values of the HGH concentration the sample can be divided into two groups; the first group reached the highest values after 20--40 min, the second one after 60--90 min. On evaluation of the curve of the mean values it appears that 2 blood samples taken 40 and 90 min after the ingestion of L-Dopa and L-Carbidopa are sufficient in order to exclude HGH deficiency. The theoretical background for HGH secretion after stimulation by L-Dopa and L-Carbidopa is discussed.
[PubMed - indexed for MEDLINE]
[HGH secretion after oral application of L-dop... [Eur J Pediatr. 1976] - PubMed - NCBI
The ingredients in 2 caps of prolactrone is comparable to what was used in the study above.
so how do you dose it? what is the ingredient profile? can't seem to find it
166mg 99% Ldopa.
333mg Green tea extract standardized for 50% EGCG. Or 166mg EGCG as this is the important portion.
Dosing for Prolactin inhibition is 1-3 caps.
For HGH increase 2-3.
1 am, 1 lunch, 1 dinner is best. Not before bed though because you get a good HGH spike during sleep.
If used standalone , at the label dose, when ended I'll need a PCT with SERM?
You don't need a pct with this stand-alone.
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I've been using formestane for years. It's my favorite AI of all time. I keep a healthy stash thinking it may be banned one day. It's only exhibits it's anabolic effects at doses approaching 800 mg/day. This makes it too expensive for anabolic purposes. There are cheaper options.
i think formestane is on the big ban bill along with every prohormoneOriginally Posted by BBB
After a cycle of testosterone, for example, your testosterone levels will be low and estrogen will be high.
Testosterone will stay low if estrogen remains elevated. Many guys use a SERM. This in turn tricks the brain into thinking estrogen is low but it does nothing to lower it.
So....after the SERM you may have elevated test levels but if you have failed to lower estrogen...guess what...test levels will drop again.
You need concurrent administration of an AI so estrogen is lowered.
In males, estrogen is made primarily from testosterone via enzymatic conversion. Test> Aromatase= estrogen.
SO, when the brain senses low estrogen, guess what happens? It sends the luetinizing hormone signal to the balls to make more testosterone so in turn more estrogen can be made. So...low estrogen = high test. High estro = low test.
THere is 0 argument for not using an AI during PCT.
One of the side effects indicated by users of Lentaron (formestane) was facial hair growth and other androgenic issues.
250mg-400mg will do the trick. Of course 800 would be more anabolic but certainly its anabolic effect is significant at half or less the mg/day.
Its like saying it only becomes a prohormone at 800mg which, is false. Even at 10mg some Formestane will be converted to hydroxytestosterone which is anabolic. After saying this there is no way to say you need 800mg to be anabolic.
Most people would have severe joint issues even at 400mg daily let alone 800.
Id be willing to see any research study proving your point. Please dont post some posts from someone else.
I can say from personal experience. But here's someone who far more knowledgable than I on the subject:
Formestane is a steroidal aromatase inhibitor, known as a suicidal inhibitor because it permanently binds to the aromatase enzyme.
Formestane was originally used as an injectable for breast cancer patients, but due to its possible androgenic effects it has largely been replaced by non-steroidial AIs in the medical community. Most of its use is now is limited to the bodybuilding community since it is available as a legal dietary supplement for reducing estrogen and increasing testosterone production.
Although formestane can effectively reduce estrogen through oral consumption, its low oral bioavailability has lead to the development of several transdermal based products (which appear to offer higher efficacy at a lower dose).
Relative to 6-oxo and ATD, Formestane is a more potent aromatase inhibitor, which appears to effectively reduce natural estrogen levels by as much as 50% within several days (while higher doses may further suppress estrogen). Because of formestanes potent ability at reducing estrogen it will tend to reduce HDL levels, while increasing LDL levels, thus harming the cholesterol profile. For this reason, it is recommended to limit cycles of formestane to 8 weeks max.
Because formestane also has a strong affinity for the 5a-reductase enzyme it will reduce DHT levels in the body by effectively competing with testosterone for the 5a-reductase.
Formestane converts to the active androgen 4-hydroxytestosterone which has about half the anabolic potency, and about 25% of the androgenic potency as testosterone. This would suggest that fairly high doses of formestane (800-1000mg/day) could lead to some level of anabolic enhancement (although the amount required for this would surely lead to undesirably low estrogen levels).
Formestane is successfully used as a standalone during re-composition cycles to help reduce “bloat” and fat storage. It can also be used as an anti-estrogen to counter aromatization of aromatizing steroids.
All I asked was to not copy past someone elses post.
Just because he claims to know doesnt mean ****. Sorry.
Also there several serious errors in that post.
Dont trust everything you read on the internet.
1. Orally Formestane is actually comparable or even better than transdermal. I know everything thinks otherwise but its just not so.
This is primarily because Formestane is not soluble in anything non toxic. This means the formestane transdermals are suspensions or partial solutions at best. Trust me. I know because I own a company that manufactures transdermal formestane. This isnt guesstimates. I know for a fact because I work with it every day and we have run tons of testing and bloodwork. I also use both the transdermal and the oral. 300mg oral formestane is awesome and preferable. Its an expensive option though. We use the transdermal because everyone likes it and the general public prefers it because previous oral versions have been severely underdosed. Primordial performance made an oral form....25mg doses. 5 days worth at usable doses per bottle...who would want that?
The problem is most companies who sold oral form sold it in 25mg doses.....pretty much worthless. 25-50mg no. 250-300 yes.
As an AI its great in the 100mg -250mg range. Just like Formeron which is transdermal.
2. There has never ever once been any proof that Formestane lowers HDL. UNtil I see a research study this is also bull**** some guy just made up.
This doesnt happen with all AI's for the record and even for those which it did happen it was at super high doses over years of administration.
3. Formestane does not even close to have a strong affinity for 5-ar. Its very weak. I have discussed this at length with patrick arnold and again until I see proof otherwise this is false as well. It has SOME affinity but certainly not strong.
Also...ever take 800-1000mg formestane? I have. Your fuking joints hurt beyond the point of being able to train.
The author here is just making up whatever he thinks is right with no basis for truth.
People should not be able to speak unless they are telling the truth.