I'm experienced with "real" stuff as well. Of course most orals are more toxic than injectables but you should compare "real" oral steroids like stanozolol, oxymetholone, oxandrolone, dianabol, turinabol, methyltestosterone and methyltrienolone to oral designer steroids.
Of course some designer steroids like superdrol and methyl-1-testosterone are very toxic to liver and not healthy to other organs either. But then there are compounds like Epi which is much less toxic than methyltrienolone (that's straight out poison by the way), oxymetholone and stanozolol.
And TR3N and TR3ST - they're both nonmethyls (not 17a-alkylated) and they don't stress liver nearly as much as traditional 17a-alkylated oral steroids. I'm sure you can find several studies and blood works to prove that. Here is one:
J. Androl., vol. 31(5) pp. 472-81Effects of synthetic androgens on liver function using the rabbit as a modelHild, SA; Attardi, BJ; Koduri, S; Till, BA; Reel, JRThe objective of this study was to determine whether the rabbit was a suitable model to test new synthetic androgens for potential liver toxicity within a short dosing interval. Adult male rabbits were dosed orally daily on days 0-13 with 17α-methyltestosterone (MT) as a positive control and testosterone (T) as a negative control to validate this model. Synthetic androgens tested were: 7α-methyl-19-nortestosterone (MENT), dimethandrolone-undecanoate (DMAU), and 11β-methyl-19-nortestosterone-17β-dodecylcarbonate (11β-MNTDC). Serum levels of alanine aminotransferase (ALT), aspartate aminotransferase (AST), gamma glutamyl transpeptidase (GGT), and sorbitol dehydrogenase (SDH), as well as clearance of intravenous injected bromsulfonphthalein (BSP) from serum on days 0, 7, and 14, were determined. As expected, T (10 mg/kg/d) did not adversely affect BSP retention or serum liver enzymes. MT (10 mg/kg/d) increased BSP retention, and AST, ALT, GGT, and SDH levels, indicating that this model could detect androgens known to be hepatotoxic. DMAU and MENT (10 mg/kg/d) increased BSP retention and all 4 serum liver enzymes as well, but the effects were less than those observed with MT at the same dose. All parameters returned to baseline 2 weeks after cessation of dosing. 11β-MNTDC at 10 mg/kg/d did not have an effect on BSP retention or liver enzymes, but a slight increase in serum GGT levels was observed in rabbits treated with 25 mg/kg/d. For the androgens that exhibited liver toxicity at 10 mg/kg/d (MT, DMAU, and MENT), a no-observed-effect level of 1 mg/kg/d was established. Overall ranking of the synthetic androgens from most to least hepatotoxic on the basis of percent BSP retention was: MT & DMAU > MENT > 11β-MNTDC. Hence, the rabbit appears to be a promising model for detection of potential liver toxicity by synthetic androgens using BSP clearance and serum liver enzyme levels as early indicators of injury.
Of course it was done with rabbits but it may be a good indicator for humans as well. Many of you won't even bother to read through it so I bolded the most important part. Ment is trestolone and MT is methyltestosterone.
If you're smart enough, you can combine the best of both worlds. My next cycle will be test, trest and methylated designer steroids.
